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Part 1 - Basic Science and General Principles

Published online by Cambridge University Press:  29 May 2020

Edited by
Peter M. Haddad  and
David J. Nutt
Peter M. Haddad
Affiliation:
Hamad Medical Corporation, Qatar
David J. Nutt
Affiliation:
Centre for Neuropsychopharmacology, Division of Psychiatry, Department of Brain Sciences, Imperial College London
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Seminars in Clinical Psychopharmacology , pp. 1 - 226
Publisher: Cambridge University Press
Print publication year: 2020

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References

Primary Sources

Abrahamsson, T, Chou, CYC, Li, SY, et al. (2017). Differential regulation and evoked spontaneous release by presynaptic NMDA receptors. Neuron, 96, 839–855.Google Scholar
Berger, M, Gray, JA, Roth, BL (2009). The expanded biology of serotonin. Ann Rev Med, 60, 355–366.Google Scholar
Chandler, CM, Overton, JS, Rüedi-Bettschen, D, Platt, DM (2018). GABA receptor subtype mechanisms and abuse-related effects of ethanol: genetic and pharmacological evidence. Handb Exp Pharmacol, 248, 3–27. doi:10.1007/164_2017_80.Google Scholar
Chen, XX, Zhang, JH, Pan, BH, et al. (2017). The atypical antipsychotic olanzapine causes weight gain by targeting 5HT2 receptors. Life Sci, 187, 64–73.Google Scholar
Cortes-Altamirano, JL, Olmos-Hernandez, A, Bonilla-Jaime, H, et al. (2017). Review: 5HT1, 5HT2, 5HT3 and 5HT7 receptors and their role in the modulation of pain response in the central nervous system. Curr Neuropharmacol, 16, 210–221. doi:10.2174/1570159x15666170911121027.Google Scholar
Di Menna, L, Joffe, ME, Iacovelli, L, et al. (2018). Functional partnership between mGlu3 and mGlu5 metabotropic glutamate receptors in the central nervous system. Neuropharmacology, 128, 301–313.Google Scholar
Doly, S, Quentin, E, Eddine, R, et al. (2017). Serotonin 2B receptors in mesoaccumbens dopaminergic pathway regulate dopaminergic responses. J Neurosci, 37, 10372–10388.Google Scholar
Glasgow, NG, Povysheva, NV, Azoleifa, AM, Johnson, JW (2017). Memantine and ketamine differentially alter NMDA receptor desensitization. J Neurosci, 37, 9686–9704.Google Scholar
Huang, X, Yang, J, Yang, S, et al. (2017). Role of tandospirone, a 5HT1A receptor partial agonist, in the treatment of CNS disorders and the underlying mechanisms. Oncotarget, 8, 102705–102720.Google Scholar
Kaushik, V, Smith, ST, Mikobi, E, Kaji, MA (2018). Acetylcholinesterase inhibitors: beneficial effects on co-morbidities in patients with Alzheimer’s disease. Am J Alzheimers Dis Other Dement, 33, 73–85.Google Scholar
Li, D-J, Fu, H, Tong, B, et al. (2018). Cholinergic anti-inflammatory pathway inhibits neointimal hyperplasia by suppressing inflammation and oxidative stress. Redox Biol, 15, 22–33.Google Scholar
Makera, C, Dondio, G, Braida, D, et al. (2018). Effects of the antidepressant mirtazepine and zinc on nicotinic acetylcholine receptors. Eur J Pharmacol, 820, 265–273.Google Scholar
Niswender, CM, Conn, PJ (2010). Metabotropic glutamate receptors: physiology, pharmacology and disease. Ann Rev Pharmacol Toxicol, 50, 295–322.Google Scholar
Overden, ES, McGregor, NW, Emsley, RA, Warnick, L (2018). DNA methylation and antipsychotic treatment in schizophrenia: progress and future directions. Prog Neuropsychopharmacol Biol Psychiatry, 81, 38–49.Google Scholar
Palma, E, Ruffolo, G, Cifelli, P, et al. (2017). Modulation of GABA-A receptors in the treatment of epilepsy. Curr Pharm Des, 23, 5563–5568. doi.2174/1381612823666170809100230.Google Scholar
Pytliak, M, Vargova, V, Mechirova, V, Felsoci, M (2011). Serotonin receptors – from molecular biology to clinical applications. Physiol Rev, 60, 15–25.Google Scholar
Viana, GSDB, Xanvier, CC, Costa, RD, Neves, KRT (2018). The monoaminergic pathways and inhibition of monoamine transporters interfere with the antidepressant-like behavior of ketamine. IBRO Rep, 4, 7–13.Google Scholar
Vizi, ES, Lendvai, B (1999). Modulatory role of presynaptic nicotinic receptors in synaptic and non synaptic chemical communication in the central nervous system. Brain Res Rev, 30, 219–235.Google Scholar
Williams, DJ, Sidaway, P, Cunnane, TV, Brain, KL (2011). Mechanisms involved in nicotinic acetylcholine receptor induced neurotransmitter release from sympathetic nerve terminals. PLoS One, 6, e29209. doi:1371/journal.pone 0029209.Google Scholar
Xiao, X, Shang, X, Zhai, B, et al. (2018). Nicotine alleviates chronic stress induced anxiety in patients with Alzheimer’s disease. Neurochem Int, 114, 58–70.Google Scholar

Secondary Sources

Ahmadi-Soleimani, SM, Azizi, H, Gompf, HS, Semnanian, S (2017). Role of orexin type-1 receptors in the paragiganto-coerulear modulation of opioid withdrawal and tolerance; a site specific focus. Neuropharmacology, 126, 25–37.Google Scholar
Andrabi, SS, Parvez, S, Tabassum, H (2017). Neurosteroids and ischaemic stroke: progesterone as a promising agent in reducing brain injury in ischemic stroke. J Environ Pathol Toxicol Oncol, 36, 191–205.Google Scholar
Balthazart, J, Choleris, E, Remage-Healey, L (2018). Steroids and the brain: 50 years of research, conceptual shifts and the ascent of non-classical and membrane initiated actions. Horm Behav, 99, 1–8.Google Scholar
Bodnar, RJ (2017). Endogenous opiates and behavior: 2015. Peptides, 88, 126–188.Google Scholar
D’Souza, DC, Sewell, RA, Ranganathan, M (2009). Cannabis and psychosis/schizophrenia: human studies. Eur Arch Psychiatry Clin Neurosci, 259, 413–431.Google Scholar
Fantegrosse, WE, Wilson, CD, Berquist, MD (2018). Pro-psychotic effects of synthetic cannabinoids: interactions with central dopamine, serotonin and glutamate receptors. Drug Metab Rev, 50, 65–78. doi:10.1080/03602532.2018.1428343.Google Scholar
Grund, T, Goyon, S, Li, Y, et al. (2007). Neuropeptide S activates paraventricular oxytocin neurons to induce anxiolysis. J Neurosci, 37, 12214–12225.Google Scholar
Henry, MS, Gendron, L, Tremblay, ME, Drolet, G (2017). Enkephalins: endogenous analgesics with an emerging role in stress resilience. Neural Plast, 2017, 1546125.Google Scholar
Smith, AS, Tabbaa, M, Lei, K, et al. (2016). Local oxytocin tempers anxiety by activating GABAA receptors in the hypothalamic paraventricular nucleus. Psychoneuroendocrinology, 63, 50–58.Google Scholar
Starwicz, K, Finn, DP (2017). Cannabinoids and pain: sites and mechanisms of action. Adv Pharmacol, 80, 437–475.Google Scholar

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