Book contents
- Frontmatter
- Contents
- Contributors
- Preface
- Herbicide use and invention
- Herbicides interacting with photosystem II
- Herbicides interacting with photosystem I
- Carotenoids and chlorophylls: herbicidal inhibition of pigment biosynthesis
- Herbicides inhibiting lipid synthesis
- The shikimate pathway as a target for herbicides
- Herbicides that inhibit the biosynthesis of branched chain amino acids
- Glutamine synthetase and its inhibition
- Metabolism of herbicides – detoxification as a basis of selectivity
- Bioactivated herbicides
- Mechanisms involved in the evolution of herbicide resistance in weeds
- Conferring herbicide resistance on susceptible crops
- Herbicide glossary
- Herbicide index
- General index
Mechanisms involved in the evolution of herbicide resistance in weeds
Published online by Cambridge University Press: 04 August 2010
- Frontmatter
- Contents
- Contributors
- Preface
- Herbicide use and invention
- Herbicides interacting with photosystem II
- Herbicides interacting with photosystem I
- Carotenoids and chlorophylls: herbicidal inhibition of pigment biosynthesis
- Herbicides inhibiting lipid synthesis
- The shikimate pathway as a target for herbicides
- Herbicides that inhibit the biosynthesis of branched chain amino acids
- Glutamine synthetase and its inhibition
- Metabolism of herbicides – detoxification as a basis of selectivity
- Bioactivated herbicides
- Mechanisms involved in the evolution of herbicide resistance in weeds
- Conferring herbicide resistance on susceptible crops
- Herbicide glossary
- Herbicide index
- General index
Summary
Introduction
Occurrence and spread of resistance to triazine herbicides
The first incidences of resistance in weed species to triazine herbicides occurred in 1968 in the State of Washington USA. During the 1970s and 1980s, notably in North America and mainly western Europe, and to a lesser extent in Israel in the 1980s there has been an irregular but relatively steady addition to the occurrences of new species becoming resistant mainly to triazine herbicides (but also to some others). Figure 1 gives an indication of the total number of species in different countries which are occurrences of resistance not previously recorded. Worldwide 49 species of 33 genera have become resistant to triazine herbicides (Le Baron pers. comm. 1987) and a further 11 species have evolved resistance to other herbicides.
A record of the occurrence of a herbicide-resistant biotype will often conceal the fact that numerous populations have independently evolved in many different locations over a period of just a few years. For example, in Hungary, Amaranthus retroflexus resistant to-s-triazines occurred in scores of locations and 75% of the maize growing area has become infested by the resistant biotype (Hartmann, 1979). In the USA, evolution of resistance to s-triazines in A. hybridus first occurred in Maryland in 1972 but between 1976 and 1982 there were numerous reports of resistant populations occurring throughout Virginia, New York, Delaware, Pennsylvania, Massachusetts, West Virginia and Illinois (Le Baron & Gressel, 1982; Le Baron pers. comm., 1987).
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- Information
- Herbicides and Plant Metabolism , pp. 211 - 236Publisher: Cambridge University PressPrint publication year: 1990