Book contents
- Frontmatter
- Contents
- Contributors
- Preface
- Acknowledgements
- Part I Introduction
- Part II Clinical manifestations and management
- 4 Soft tissue patterns in acute electric burns
- 5 The spectrum of electrical injuries
- 6 Electrical trauma: pathophysiology and clinical management
- 7 Acute and delayed neurological sequelae of electrical injury
- 8 Paediatric electrical burns
- 9 Surgical technique for salvage of elecrically damaged tissue
- Part III Tissue responses
- Part IV Biophysical mechanisms of cellular injury
- Index
6 - Electrical trauma: pathophysiology and clinical management
from Part II - Clinical manifestations and management
Published online by Cambridge University Press: 08 April 2010
- Frontmatter
- Contents
- Contributors
- Preface
- Acknowledgements
- Part I Introduction
- Part II Clinical manifestations and management
- 4 Soft tissue patterns in acute electric burns
- 5 The spectrum of electrical injuries
- 6 Electrical trauma: pathophysiology and clinical management
- 7 Acute and delayed neurological sequelae of electrical injury
- 8 Paediatric electrical burns
- 9 Surgical technique for salvage of elecrically damaged tissue
- Part III Tissue responses
- Part IV Biophysical mechanisms of cellular injury
- Index
Summary
Electrical burn injuries account for less than 5% of admissions to major burn centres. However, the injury is much more complex than a skin burn and the morbidity and mortality rate is considerably higher. The mortality rate ranges from 3–15% with about 1000 deaths attributed to electrical current in the US each year. More than 90% of injuries occur in males, most between ages 20 and 34.
Electrical current tissue damage
Pathophysiology
The severity of injury to tissues is dependent on the amperage, i.e. the actual amount of current, passing through the tissues. It is impossible to know the amperage because of the variability of resistance and exposure time at the accident, but one can infer amperage from the voltage of the source at least as to high or low. A low-voltage source is capable of producing major cardiopulmonary complications and death if a sufficient current passes through the body (Table 6.1).
A high-tension source is usually required to produce the severe tissue necrosis characteristically seen along the path of the current. The damage is caused by both heat production and direct current damage. The initial resistance to flow of current, namely, skin or clothing, is overcome by the heat generated from the high voltage, and subsequent tissue necrosis occurs with continued contact. A dry hand may have sufficient resistance to avoid passage of current from a low-voltage source over a short time period.
- Type
- Chapter
- Information
- Electrical TraumaThe Pathophysiology, Manifestations and Clinical Management, pp. 122 - 132Publisher: Cambridge University PressPrint publication year: 1992
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