Book contents
- Frontmatter
- Contents
- List of contributors
- List of abbreviations
- Preface
- Section 1 Bilateral Predominantly Symmetric Abnormalities
- Section 2 Sellar, Perisellar and Midline Lesions
- 38 Rathke's Cleft Cyst
- 39 Pituitary Microadenoma
- 40 Lymphocytic Hypophysitis
- 41 Pituitary Macroadenoma
- 42 Ectopic Posterior Pituitary Lobe
- 43 Langerhans Cell Histiocytosis
- 44 Craniopharyngioma
- 45 Hypothalamic Hamartoma
- 46 Optic Glioma
- 47 Perisellar Meningioma
- 48 Hemangioma of the Cavernous Sinus
- 49 Tolosa–Hunt Syndrome
- 50 Carotid-Cavernous Sinus Fistula
- 51 Perisellar Aneurysm
- 52 Chordoma
- 53 Chondrosarcoma
- 54 Colloid Cyst
- 55 Aqueductal Stenosis
- 56 Progressive Supranuclear Palsy (PSP)
- 57 Joubert Syndrome
- 58 Rhombencephalosynapsis
- 59 Multiple System Atrophy (MSA)
- 60 Maple Syrup Urine Disease (MSUD)
- 61 Chiari 2 Malformation
- 62 Tectal Glioma
- 63 Brainstem Glioma
- 64 Duret Hemorrhage
- 65 Hypertrophic Olivary Degeneration
- 66 Osmotic Myelinolysis
- 67 Germinoma
- 68 Pineoblastoma
- 69 Pineal Cyst
- 70 Vein of Galen Aneurysmal Malformation (VGAM)
- 71 Corpus Callosum Dysgenesis
- 72 Septo-Optic Dysplasia
- 73 Holoprosencephaly
- 74 Atretic Parietal Encephalocele
- 75 Dermoid Cyst
- 76 Lipoma
- Section 3 Parenchymal Defects or Abnormal Volume
- Section 4 Abnormalities Without Significant Mass Effect
- Section 5 Primarily Extra-Axial Focal Space-Occupying Lesions
- Section 6 Primarily Intra-Axial Masses
- Section 7 Intracranial Calcifications
- Index
- References
48 - Hemangioma of the Cavernous Sinus
from Section 2 - Sellar, Perisellar and Midline Lesions
Published online by Cambridge University Press: 05 August 2013
- Frontmatter
- Contents
- List of contributors
- List of abbreviations
- Preface
- Section 1 Bilateral Predominantly Symmetric Abnormalities
- Section 2 Sellar, Perisellar and Midline Lesions
- 38 Rathke's Cleft Cyst
- 39 Pituitary Microadenoma
- 40 Lymphocytic Hypophysitis
- 41 Pituitary Macroadenoma
- 42 Ectopic Posterior Pituitary Lobe
- 43 Langerhans Cell Histiocytosis
- 44 Craniopharyngioma
- 45 Hypothalamic Hamartoma
- 46 Optic Glioma
- 47 Perisellar Meningioma
- 48 Hemangioma of the Cavernous Sinus
- 49 Tolosa–Hunt Syndrome
- 50 Carotid-Cavernous Sinus Fistula
- 51 Perisellar Aneurysm
- 52 Chordoma
- 53 Chondrosarcoma
- 54 Colloid Cyst
- 55 Aqueductal Stenosis
- 56 Progressive Supranuclear Palsy (PSP)
- 57 Joubert Syndrome
- 58 Rhombencephalosynapsis
- 59 Multiple System Atrophy (MSA)
- 60 Maple Syrup Urine Disease (MSUD)
- 61 Chiari 2 Malformation
- 62 Tectal Glioma
- 63 Brainstem Glioma
- 64 Duret Hemorrhage
- 65 Hypertrophic Olivary Degeneration
- 66 Osmotic Myelinolysis
- 67 Germinoma
- 68 Pineoblastoma
- 69 Pineal Cyst
- 70 Vein of Galen Aneurysmal Malformation (VGAM)
- 71 Corpus Callosum Dysgenesis
- 72 Septo-Optic Dysplasia
- 73 Holoprosencephaly
- 74 Atretic Parietal Encephalocele
- 75 Dermoid Cyst
- 76 Lipoma
- Section 3 Parenchymal Defects or Abnormal Volume
- Section 4 Abnormalities Without Significant Mass Effect
- Section 5 Primarily Extra-Axial Focal Space-Occupying Lesions
- Section 6 Primarily Intra-Axial Masses
- Section 7 Intracranial Calcifications
- Index
- References
Summary
Specific Imaging Findings
Hemangioma (cavernous hemangioma, cavernous malformation) of the cavernous sinus shows characteristically marked T2 hyperintensity, approaching that of CSF. The tumors are typically homogenous and well-delineated, hypointense on T1-weighted images and with avid post-contrast enhancement. Enhancement may be homogenous or heterogenous, depending on the histologic type. Reflecting their large extracellular spaces, these benign neoplasms also show very high diffusion of water molecules, being almost as bright as CSF on ADC maps. Hemangiomas are hypodense on CT, remodeling of the adjacent bone may be present, without destruction or invasion. Similar to hemangiomas in other parts of the body, these vascular tumors demonstrate progressive filling in with contrast on dynamic and delayed MRI and CT images. Their very early enhancement following contrast bolus has a prominent tumor blush appearance, which may be appreciated on cranial CTA studies.
Pertinent Clinical Information
Cavernous sinus hemangioma may possibly be an incidental finding; however, they usually present with diplopia (extraocular muscle palsy, primarily oculomotor nerve), facial numbness, visual loss and headache. Optimal management is not clear at this time – total microsurgical removal is effective but may be extremely difficult and carries the risk of cranial nerve deficits; low-dose radiosurgery appears to lead to significant tumor shrinkage and clinical improvement. While radiosurgery may also be used following incomplete tumor excision, it is likely to become the treatment modality of choice.
- Type
- Chapter
- Information
- Brain Imaging with MRI and CTAn Image Pattern Approach, pp. 99 - 100Publisher: Cambridge University PressPrint publication year: 2012