Following six consecutive annual applications of imazaquin in combination with trifluralin or pendimethalin to several soybean fields on the Delmarva Peninsula, unacceptable smooth pigweed control was observed. Field and greenhouse studies were conducted to determine if this population of smooth pigweed was resistant to imazaquin and other herbicides. In field research, imazaquin and imazethapyr gave complete control of the susceptible (S) population while providing no control of the resistant (R) population; pyrithiobac controlled 99 and 90% of the R and S populations, respectively. Pendimethalin, metribuzin, MON-12000, and flumiclorac gave less than 75% control of both S and R populations. Chlorimuron, primisulfuron, CGA-152005, and lactofen gave above 75% control, and thifensulfuron and nicosulfuron gave above 90% control of both S and R populations. Seeds were collected from the R and S smooth pigweed populations for research in the greenhouse. Greenhouse studies confirmed high levels of resistance to imazaquin and imazethapyr and low levels of cross-resistance to rimsulfuron and chlorimuron in the R population. Susceptibility of the R population to nicosulfuron, thifensulfuron, pyrithiobac, and pendimethalin was comparable to that of the S population.

Greenhouse and field experiments were conducted to evaluate the potential for antagonistic or synergistic interactions from tank mixtures of glyphosate plus a selective herbicide applied postemergence. In the greenhouse, glyphosate at 420 g ae ha−1 plus 28% liquid urea-ammonium nitrate (28% UAN) provided at least 89% control of common lambsquarters and common ragweed. Glyphosate at 1,680 g ha−1 plus 28% UAN provided less than 81% control of velvetleaf and less than 75% control of ivyleaf morningglory. Tank mixing bentazon at 1,120 g ai ha−1 with glyphosate at 420 g ha−1 synergistically increased control of velvetleaf. Tank mixtures of glyphosate plus a selective herbicide were predominately additive in control of common lambsquarters, common ragweed, and velvetleaf. Several tank mix combinations of chlorimuron or imazethapyr plus glyphosate plus 28% UAN were antagonistic in control of ivyleaf morningglory. In the field, glyphosate at 840 g ha−1 plus 28% UAN provided at least 88% control of common lambsquarters and velvetleaf in 1994. However, glyphosate at 840 g ha−1 plus 28% UAN provided only 60% control of velvetleaf in 1995. Tank-mixing bentazon or CGA-248757 with glyphosate at 420 g ha−1 increased velvetleaf and common lambsquarters control in 1995. In general, adding chlorimuron, imazethapyr, or thifensulfuron to glyphosate plus 28% UAN did not increase control of common lambsquarters or velvetleaf. Tank mixing imazethapyr with glyphosate plus 28% UAN antagonized velvetleaf control in 1994 and in 1995. The tank mixture of thifensulfuron at 2 g ha−1 plus glyphosate at 420 g ha−1 plus 28% UAN increased soybean injury in the field in 1994. However, tank mixing chlorimuron, imazethapyr, or thifensulfuron with glyphosate plus 28% UAN did not increase soybean injury in the greenhouse or in the field in 1995.

Knowledge of how reduction in the rate of herbicide application or rotation of their mode of action influences weed growth will provide insight into how successful these practices will be in an integrated weed management program. Field experiments were conducted in 1996 and 1997 to quantify velvetleaf growth response to three postemergence herbicides, each with a different mode of action. A monoculture of velvetleaf was treated with halosulfuron, dicamba, and flumiclorac at 0, 0.10, 0.25, 0.50, 0.75, and 1.0 × the labeled rate for weed control in corn. Percent plant mortality increased with rate of application; the greatest mortality occurred in flumiclorac treatments in 1996 and in halosulfuron and flumiclorac treatments in 1997. Growth rate temporarily decreased as application rate increased. Maximum height decreased as rate of application increased, with the dicamba treatment resulting in the greatest (27%) reduction. Early-season leaf area index decreased with increasing rate of application, the greatest reduction occurring with halosulfuron (1997) and flumiclorac (1996 and 1997) treatments. The number of leaves produced per plant was temporarily reduced by all treatments, but treatment with dicamba later resulted in larger numbers of small leaves. The number of velvetleaf seed capsules produced per surviving plant was not reduced by any treatment, but the number of capsules per square meter was reduced by the 0.5 × rate of flumiclorac (1996) and the 0.5- and 1.0 × rates of halosulfuron (1997). Research is needed to evaluate whether the temporary suspension of velvetleaf growth after herbicide treatment is sufficient to prohibit crop yield reduction and velvetleaf capsule production.

Greenhouse and laboratory studies were conducted to confirm and quantify glyphosate resistance, quantify pyrithiobac resistance, and investigate interaction between flumiclorac and glyphosate mixtures on control of Palmer amaranth from Mississippi. The GR50 (herbicide dose required to cause a 50% reduction in plant growth) values for two glyphosate-resistant biotypes, C1B1 and T4B1, and a glyphosate-susceptible (GS) biotype were 1.52, 1.3, and 0.09 kg ae ha−1 glyphosate, respectively. This indicated that the C1B1 and T4B1 biotypes were 17- and 14-fold resistant to glyphosate, respectively, compared with the GS biotype. The C1B1 and T4B1 biotypes were also resistant to pyrithiobac, an acetolactate synthase (ALS) inhibitor, with GR50 values of 0.06 and 0.07 kg ai ha−1, respectively. This indicated that the C1B1 and T4B1 biotypes were 7- and 8-fold, respectively, more resistant to pyrithiobac compared with the GS biotype, which had a GR50 value of 0.009 kg ha−1. Flumiclorac was antagonistic to glyphosate by reducing glyphosate translocation. The C1B1 and T4B1 absorbed less glyphosate 48 h after treatment (HAT) compared with the GS biotype. The majority of the translocated glyphosate accumulated in the shoot above the treated leaf (that contains the apical meristem) in the GS biotype and in the shoot below the treated leaf in the resistant biotypes, C1B1 and T4B1, by 48 HAT. The C1B1 biotype accumulated negligible shikimate levels, whereas the T4B1 and GS biotypes recorded elevated levels of shikimate. Metabolism of glyphosate to aminomethylphosphonic acid was not detected in either of the resistant biotypes or the susceptible GS biotype. The above results confirm multiple resistance to glyphosate and pyrithiobac in Palmer amaranth biotypes from Mississippi and indicate that resistance to glyphosate is partly due to reduced absorption and translocation of glyphosate.

Field studies were conducted from 1997 to 1999 to evaluate the influence of broadleaf herbicides on double-crop glyphosate-resistant soybean injury and yield at Belleville, IL. Diphenylether herbicides applied postemergence (POST) caused 10 to 48% necrosis 7 days after treatment (DAT). POST herbicides caused 0 to 8%, 8 to 37%, and 0 to 12% height reduction 7 DAT in 1997, 1998, and 1999, respectively. In most instances, diphenylether herbicides did not delay soybean maturity, whereas imazethapyr applied POST delayed soybean maturity in 1998 and 1999. Acifluorfen plus bentazon reduced soybean height at maturity in 1997 and 1998, whereas lactofen, imazethapyr, and imazamox reduced height only in 1998. Diphenylether herbicides did not reduce grain yield when compared to the no-herbicide hand-weeded treatment, but imazamox reduced yield by 18%. Furthermore, the soil herbicides applied preemergence and diphenylether herbicides applied POST did not reduce grain yield when compared to glyphosate alone.

Field trials were conducted at two sites in both 1997 and 1998 to evaluate soybean response and weed control with glufosinate alone or combined with quizalofop, lactofen, imazethapyr, flumiclorac, or bentazon plus acifluorfen in narrow-row, glufosinate-resistant soybean. Soybean injury ranged from 0 to 21% at 2 wk after treatment (WAT) and from 0 to 5% by 4 WAT. Glufosinate alone at 0.29 and 0.4 kg ai/ha controlled velvetleaf, common waterhemp, common ragweed, morningglory species, and giant foxtail greater than 85% in all studies. Mixtures containing glufosinate and other herbicides controlled these species greater than 81% but did not improve control over glufosinate alone. Estimates of weed biomass closely reflected visual control evaluations. However, giant foxtail biomass was higher for mixtures of glufosinate plus lactofen, flumiclorac, or bentazon and acifluorfen, indicating possible antagonism of glufosinate activity. At both locations, soybean yields were similar among most treatments, but that of the glufosinate plus lactofen treatment was lower when compared with other treatments. Additional trials evaluated soybean response and weed control with a preemergence herbicide followed by glufosinate postemergence (POST), glufosinate applied once or twice POST, and mixtures of glufosinate plus imazethapyr or flumiclorac POST in wide-row soybean. Glufosinate applied twice controlled common waterhemp, morningglory species, prickly sida, common cocklebur, and giant foxtail up to 39% greater than did glufosinate applied once. The addition of imazethapyr, but not flumiclorac, to glufosinate improved weed control when compared with glufosinate alone.

Cotton boll weevil has been eradicated from much of the U.S. Cotton Belt. After eradication, a containment program is necessary to detect and destroy reintroduced boll weevils. Crops other than cotton are not monitored for boll weevil, hence fruit on volunteer glyphosate-resistant (GR) cotton in GR soybean could provide oviposition sites for boll weevils and allow the insects to build up undetected. An experiment was conducted at five locations to evaluate control of GR cotton and reduction in cotton fruit production by herbicides commonly used on GR soybean. Cotton control by preemergence (PRE) or postemergence (POST) herbicides alone was inconsistent across locations. Flumetsulam at 45 g ai/ha, imazaquin at 137 g ai/ha, and metribuzin at 360 g ai/ha plus chlorimuron at 60 g ai/ha applied PRE controlled cotton 55 to 100% and reduced cotton fruit production 84 to 100%. Sulfentrazone at 167 g ai/ha plus chlorimuron at 34 g/ha PRE controlled cotton 50 to 91% and reduced fruit 48 to 98%. Metribuzin PRE at 420 g/ha controlled cotton 23 to 97% and reduced fruit 32 to 100%. Flumiclorac at 30 g ai/ha, 2,4-DB dimethylamine salt at 35 g ae/ha, chlorimuron at 12 g ai/ha, and the sodium salt of fomesafen at 420 g ai/ha mixed with glyphosate and applied POST controlled cotton 48 to 100% and reduced fruit production 67 to 100%. Cloransulam at 12 or 18 g ai/ha controlled cotton 3 to 66% and reduced fruit production 5 to 85%. Cotton control and fruit reduction were greatest and most consistent with sequential applications of metribuzin plus chlorimuron PRE followed by chlorimuron, flumiclorac, fomesafen, or 2,4-DB POST. These treatments controlled cotton at least 95% at all locations. Cotton fruit was totally eliminated at three locations and reduced at least 97% at a fourth location.

Field and greenhouse studies were conducted to evaluate herbicides in pumpkin. Field experiments at three Illinois locations determined weed control and crop injury from clomazone, dimethenamid, ethalfluralin, sulfentrazone, imazamox, RPA 201772, flumiclorac, and halosulfuron applied preemergence. Clomazone plus sulfentrazone controlled redroot pigweed 78 to 99%, ivyleaf morningglory 80 to 97%, common lambsquarters 97%, common purslane 84 to 99%, and velvetleaf 55 to 99%. Imazamox plus clomazone commonly provided more consistent broadleaf weed control than did ethalfluralin plus clomazone. RPA 201772 when used on sandy soils killed the pumpkin. Sulfentrazone initially caused chlorosis and necrosis on pumpkin seedlings when soil organic matter was low (< 1%) or when soil moisture was high. However, plants recovered rapidly, and yields were not affected. In a greenhouse study pumpkin was more tolerant to clomazone plus sulfentrazone than to sulfentrazone.

The effect temperature, light intensity, time to initial light exposure, relative humidity, and the presence of dew have on CGA-248757 and flumiclorac efficacy was evaluated in laboratory trials. Increasing temperature from 10 to 40 C increased CGA-248757 and flumiclorac activity on common lambsquarters by 79 and 87%, respectively. Similarly, increasing temperature from 10 to 40 C increased CGA-248757 and flumiclorac activity on redroot pigweed by 68 and 60%, respectively. Increasing light intensity from 0 to 1,000 μmol m−2 s−1 increased CGA-248757 activity on common lambsquarters and redroot pigweed by 92 and 93%, while flumiclorac activity increased 91 and 99%. Time to initial light exposure and relative humidity did not affect CGA-248757 or flumiclorac activity on common lambsquarters and redroot pigweed. The presence of dew reduced herbicidal activity of both compounds on common lambsquarters by 5% and redroot pigweed control with CGA-248757 and flumiclorac by 21 and 20%, respectively. Field applications of CGA-248757 or flumiclorac at 6:00 A.M., 2:00 P.M., and 10:00 P.M. indicate environmental conditions at application strongly influence soybean tolerance and weed control with CGA-248757 and flumiclorac. The greatest soybean injury occurred from CGA-248757 or flumiclorac applications at 6:00 A.M. compared with applications at 2:00 P.M. or 10:00 P.M. Common lambsquarters control was greatest when CGA-248757 or flumiclorac was applied at 6:00 A.M. or 2:00 P.M. compared with 10:00 P.M. However, redroot pigweed control was greatest when CGA-248757 or flumiclorac was applied at 2:00 P.M. Application time of day did not affect velvetleaf control with either herbicide.

Field trials evaluated weed control in corn and soybean with CGA-248757 and flumiclorac. CGA-248757 and flumiclorac were applied to 5-, 45-, and 60-cm-tall velvetleaf. Visual control ratings 7 d after treatment (DAT) indicated the greatest control of velvetleaf in corn with CGA-248757 applied to velvetleaf when 5 or 45 cm tall, whereas the greatest control with flumiclorac occurred when applied to 5-cm-tall velvetleaf. However, ratings 21 DAT to 60-cm-tall velvetleaf indicated velvetleaf control with both herbicides was greatest when applied to plants 45 or 60 cm tall. CGA-248757 or flumiclorac applied postemergence (POST) following metolachlor or metolachlor plus atrazine preemergence (PRE) improved velvetleaf control compared with metolachlor plus atrazine alone. Similarly, tank mixtures of CGA-248757 or flumiclorac with 2,4-D following metolachlor PRE provided greater velvetleaf control compared with metolachlor PRE followed by 2,4-D POST. Velvetleaf control in corn was greater when CGA-248757 or flumiclorac were tank mixed with nicosulfuron plus atrazine or nicosulfuron plus dicamba. In soybean, metolachlor plus metribuzin plus clomazone PRE, metolachlor PRE followed by flumiclorac POST, and metolachlor plus metribuzin PRE followed by CGA-248757 or flumiclorac POST provided greater than 85% control of giant foxtail, common lambsquarters, redroot pigweed, and velvetleaf 56 d after POST treatments (DAPO) in 1997. Additionally, glyphosate applied late postemergence (LPOST) and tank mixtures of glyphosate with CGA-248757 or flumiclorac LPOST provided greater than 90% control of these same weed species in 1997. However, only metolachlor plus metribuzin PRE followed by CGA-248757 or flumiclorac POST and imazethapyr plus CGA-248757 or flumiclorac POST provided greater than 80% giant foxtail, common lambsquarters, redroot pigweed, and velvetleaf control 56 DAPO in 1998.

Greenhouse and laboratory studies were conducted to determine the physiological basis for CGA-248757 and flumiclorac selectivity in five plant species. CGA-248757 and flumiclorac selectively control weeds postemergence (POST) by inhibiting protoporphyrinogen oxidase (Protox). Injury symptoms from CGA-248757 and flumiclorac include rapid desiccation and necrosis similar to injury from diphenyl ether and bipyridinium herbicides. Species sensitivity to CGA-248757 and flumiclorac was evaluated by comparing the dry weight reduction from POST applications. Abutilon theophrasti was sensitive to both herbicides, Amaranthus retroflexus was more sensitive to flumiclorac than CGA-248757, Brassica kaber was sensitive to CGA-248757 but tolerant of flumiclorac, and Zea mays and Glycine max were tolerant of both herbicides. Studies evaluated CGA-248757 and flumiclorac retention, absorption, translocation, and metabolism. Enhanced herbicide metabolism contributed to the tolerance of A. retroflexus to CGA-248757 and B. kaber to flumiclorac. Decreased herbicide retention, absorption, and translocation coupled with increased metabolism contributed to Z. mays tolerance of CGA-248757 and flumiclorac. Decreased herbicide retention and increased herbicide metabolism provided G. max tolerance of both herbicides.

Broadleaf weed control with postemergence applications of CGA-248757 and flumiclorac applied alone and in tank mixtures was evaluated in greenhouse and field studies. In greenhouse studies velvetleaf, common lambsquarters, redroot pigweed, common ragweed, common cocklebur, eastern black nightshade, and wild mustard shoot weight were reduced by 50% from 0.1, 2.9, 0.9, 1.1, 0.8, 0.4, and 1.2 g/ha of CGA-248757 and 0.7, 3.0, 2.4, 3.3, 3.0, 3.4, and 74.1 g/ha of flumiclorac, respectively. Under greenhouse conditions, tank mixtures of CGA-248757 or flumiclorac with atrazine, bentazon, bromoxynil, dicamba, halosulfuron, imazethapyr, lactofen, primisulfuron plus prosulfuron, or 2,4-D increased velvetleaf, common lambsquarters, redroot pigweed, or common ragweed control 14 d after treatment (DAT) when compared with the control provided by atrazine, bentazon, bromoxynil, dicamba, halosulfuron, imazethapyr, lactofen, primisulfuron plus prosulfuron, or 2,4-D applied alone. In field studies, adding CGA-248757 or flumiclorac to atrazine, bentazon, bromoxynil, dicamba, halosulfuron, imazethapyr, lactofen, CGA-277476, primisulfuron plus prosulfuron, or 2,4-D increased crop injury 3 DAT, but there was no reduction in corn or soybean yields as a result. In corn, tank mixtures of CGA-248757 or flumiclorac plus atrazine, dicamba, or 2,4-D increased velvetleaf control compared with treatments without CGA-248757 or flumiclorac. Tank mixtures of CGA-248757 or flumiclorac with primisulfuron plus prosulfuron increased common lambsquarters control compared with primisulfuron plus prosulfuron alone. Similarly, in soybean CGA-248757 or flumiclorac tank mixtures with bentazon or CGA-277476 increased control of redroot pigweed, and tank mixtures with lactofen increased common lambsquarters control. However, season-long broadleaf control in soybean with bentazon, lactofen, and CGA-277476 tank mixtures with CGA-248757 or flumiclorac did not provide acceptable weed control. Compared with imazethapyr alone the addition of CGA-248757 and flumiclorac improved soybean yield and provided season-long eastern black nightshade, common lambsquarters, redroot pigweed, and common ragweed control.

Herbicides were evaluated for weed control and crop response in conventional and no-tillage dry pea production. Preplant or preemergence (PRE) applications of imazethapyr, sulfentrazone, flumetsulam, cloransulam, and BAY FOE 5043 + metribuzin did not show crop injury within the locations, years, and tillage systems where applied. Postemergence applications of cloransulam had crop injury in excess of 60% where applied, and injury with flumiclorac and fomesafen ranged from 0 to greater than 40% with differences in crop injury as a result of dry pea growth stage at the time of application and surfactant use. Imazamox injury was as great as 24% when applied at a more advanced dry pea growth stage and was not different from 0% when applied at an earlier growth stage. Dry pea injury with bentazon was not significant, with an exception at one of the six locations where injury was 14%. Common lambsquarters was best controlled (80 to 90%) with sulfentrazone and imazamox. Preplant and PRE applications of sulfentrazone consistently provided the greatest mayweed chamomile control across tillage systems (control ranged from 59 to 93%), whereas bentazon provided the greatest postemergence (POST) control of mayweed chamomile (control ranged from 46 to 84%). Prickly lettuce control with preplant or PRE treatments was greatest with sulfentrazone (74 to 85%), whereas the greatest POST control was with imazamox in combination with urea-ammonium nitrate solution and surfactant or bentazon and crop oil concentrate (71 to 92%). Dry pea yields with herbicide treatments were not always greater than the nontreated and were often affected by crop injury.

Common dandelion has developed into a troublesome agronomic weed for no-tillage corn producers. A postemergence herbicide application is often required to reduce common dandelion competition. Field experiments were conducted in 2002 and 2003 to evaluate 22 postemergence herbicide treatments for efficacy on established populations of common dandelion in no-tillage corn. All herbicides were applied to five- to six-collar corn at registered rates with typical adjuvants. At 28 d after treatment (DAT) the most effective treatments included glufosinate and mesotrione providing at least 76% control of common dandelion. All other herbicide treatments provided less than 40% common dandelion control 28 DAT. Common dandelion control was evaluated 56 DAT when regrowth of treated plants was observed for some herbicide treatments. AT 56 DAT, dicamba + diflufenzopyr was the most effective treatment, providing 83% control of common dandelion. In 2002, all herbicide treatments, with the exception of flumiclorac, resulted in corn yields greater than the nontreated.

Sclerotinia stem rot is an important soybean disease. An increase in phytoalexin production with herbicide treatments may reduce the incidence of this disease in soybean. Research was conducted to determine soybean response, Sclerotinia sclerotiorum lesion development, and phytoalexin production in glyphosate-resistant and -susceptible soybean cultivars treated with protoporphyrinogen oxidase–inhibiting herbicides. Necrosis of soybean leaves 7 d after postemergence application of oxyfluorfen at 17.5 g ai/ha, carfentrazone at 1.8 g ai/ha, sulfentrazone at 9.0 g ai/ha, fomesafen at 280 g ai/ha, acifluorfen at 425 g ai/ha, flumiclorac at 30 g ai/ha, CGA-248757 at 4 g ai/ha, and oxadiazon at 280 g ai/ha was equal to or less than lactofen at 70 g ai/ha. In a detached leaf bioassay, S. sclerotiorum lesion diameter was reduced by oxyfluorfen, carfentrazone, sulfentrazone, lactofen, fomesafen, flumiclorac, and oxadiazon compared with the untreated control. Furthermore, lesion diameter on untreated leaves of soybean treated with oxyfluorfen, carfentrazone, sulfentrazone, lactofen, fomesafen, acifluorfen, flumiclorac, CGA-248757, and oxadiazon was reduced compared with the untreated control. Lactofen and sulfentrazone increased leaf phytoalexin production similarly, but neither herbicide affected stem phytoalexin production compared with the untreated control. Glyphosate-resistant and near-isogenic–susceptible cultivars responded similarly when inoculated with S. sclerotiorum in the detached leaf bioassay. Glyphosate-resistant S20-B9 and P93B01 produced more phytoalexins than glyphosate-susceptible S 19-90 and P9281. Herbicide treatments may increase phytoalexin production in leaves of treated plants, but levels in the stem do not explain protection from Sclerotinia stem rot.

Injury to weeds from sublethal doses of POST herbicides may reduce the effect of weed interference on crop yield. Information on how herbicide dose influences weed mortality, growth, and seed production is needed to assess the potential benefit of applying reduced herbicide doses. Field experiments were conducted at Mead, NE, in 2001 and 2002 to quantify velvetleaf mortality, growth, and corn–velvetleaf interference in response to varying doses of three POST herbicides. Untreated velvetleaf at six densities (0, 1, 3, 6, 12, and 20 plants m−1 corn row) was grown in mixture with corn to establish a baseline corn–velvetleaf interference relationship. Treated velvetleaf at a density of 20 plants m−1 row received one of five doses of dicamba, halosulfuron, or flumiclorac. Untreated velvetleaf height, biomass, and seed capsule production were greater in 2002 than 2001 and declined with increasing velvetleaf density in both years. Corn yield was not affected by untreated velvetleaf in 2001, but yield loss increased with increasing velvetleaf density in 2002. Mortality of herbicide-treated velvetleaf was 56% greater in 2001 than 2002 and increased with increasing herbicide dose. Maximum height of treated velvetleaf was similar for all treatments in 2001 but declined with increasing herbicide dose in 2002. Biomass and seed production of treated velvetleaf varied among herbicides in 2002 and decreased with increasing dose. Corn yield was not influenced by velvetleaf in 2001, but yield loss in response to herbicide-treated velvetleaf declined with increasing herbicide dose in 2002. Results show that the assumption that weeds surviving herbicide application are as competitive as untreated weeds is incorrect. Reduction in growth and resource consumption by herbicide-damaged weeds reduced the negative effects of weeds on corn.

In a repeated multi-year study, mother potato plants were exposed to herbicides at rates that simulated off-target application, such as through tank contamination. Following exposure of mother plants to herbicides, seed from mother plants was planted in the following growing season and crop growth, yield and tuber quality were quantified. Visual injury from herbicides was observed both in the mother plant and daughter tuber growing seasons and occasional impacts on tuber yield were noted. However, an inconsistent relationship was observed for herbicide related injury and tuber yield reductions of mother potato plants with daughter tuber growth and yield. The lack of consistency in the relationship between visual potato injury in the mother plant production and adverse daughter tuber growth and yield in the following year challenges traditional crop scouting as a tool to predict off-target herbicide risk near seed potato production.