We consider the model of Deijfen, Häggström and Bagley (2004) for competing growth of two infection types in Rd, based on the Richardson model on Zd. Stochastic ball-shaped infection outbursts transmit the infection type of the center to all points of the ball that are not yet infected. Relevant parameters of the model are the initial infection configuration, the (type-dependent) growth rates, and the radius distribution of the infection outbursts. The main question is that of coexistence: Which values of the parameters allow the unbounded growth of both types with positive probability? Deijfen, Häggström and Bagley (2004) conjectured that the initial configuration is basically irrelevant for this question, and gave a proof for this under strong assumptions on the radius distribution, which, e.g. do not include the case of a deterministic radius. Here we give a proof that does not rely on these assumptions. One of the tools to be used is a slight generalization of the model with immune regions and delayed initial infection configurations.