This paper considers a model for the spread of an epidemic in a closed population whose members are in either a high-risk or a low-risk activity group. Further, members of the high-risk group may change their behaviour by entering the low-risk group. Both stochastic and deterministic models are examined. A limiting model, appropriate when there is a large number of initially susceptible individuals, is used to provide a threshold analysis. The epidemic is compared to a single group epidemic, and to suitably parametrised two-group epidemics, using a coupling method. The total size distribution and effects of changing the behaviour change rate are considered.

This paper considers a model for the spread of an epidemic in a closed, homogeneously mixing population in which new infections occur at rate βxy/(x + y), where x and y are the numbers of susceptible and infectious individuals, respectively, and β is an infection parameter. This contrasts with the standard general epidemic in which new infections occur at rate βxy. Both the deterministic and stochastic versions of the modified epidemic are analysed. The deterministic model is completely soluble. The time-dependent solution of the stochastic model is derived and the total size distribution is considered. Threshold theorems, analogous to those of Whittle (1955) and Williams (1971) for the general stochastic epidemic, are proved for the stochastic model. Comparisons are made between the modified and general epidemics. The effect of introducing variability in susceptibility into the modified epidemic is studied.

This paper is concerned with a pure death process, starting with N individuals, with death rates μ n, n = 1, 2, …, N. It is shown that the fates of distinct individuals are positively correlated if μ n/n decreases with n, and negatively correlated if μ n/n increases with n. The application of this result to the problem of variability in compartmental models is elaborated and in particular a conjecture of Faddy (1985) is settled. Further applications to well-known death processes are also briefly described.

The deterministic and stochastic models developed by Bartlett (1956) to describe the initial spatial spread of an epidemic such as measles are extended to host-vector and carrier-borne epidemics such as malaria and typhoid in which more than one type of individual is involved. If the epidemic does not die out, then the behaviour predicted by the deterministic model and an examination of the mean distributions in the stochastic model is a wave of infection spreading out from the initial source of infection.