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Aberrant inflammation in rat pregnancy leads to cardiometabolic alterations in their offspring and intrauterine growth restriction in the F2 generation – CORRIGENDUM

Published online by Cambridge University Press:  27 June 2022

Takafumi Ushida ,
Tiziana Cotechini ,
Nicole Protopapas ,
Aline Atallah ,
Charlotte Collyer ,
Alexa J. Toews ,
Shannyn K. Macdonald-Goodfellow ,
M. Yat Tse ,
Louise M. Winn  and
Stephen C. Pang
...Show all authors
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Abstract

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Type
Corrigendum
Information
Journal of Developmental Origins of Health and Disease , Volume 13 , Issue 6 , December 2022 , pp. 816
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Creative Commons
Creative Common License - CCCreative Common License - BYCreative Common License - NCCreative Common License - ND
This is an Open Access article, distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives licence (https://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is unaltered and is properly cited. The written permission of Cambridge University Press must be obtained for commercial re-use or in order to create a derivative work.
Copyright
© The Author(s), 2022. Published by Cambridge University Press in association with International Society for Developmental Origins of Health and Disease

The authors regret the inclusion of an error in the above article title.

The title of the article mistakenly read; Aberrant inflammation in rat pregnancy leads to cardiometabolic alterations in their offspring and intrauterine growth restriction in the F2 generation.

The correct title should read; Aberrant inflammation in rat pregnancy leads to cardiometabolic alterations in the offspring and intrauterine growth restriction in the F2 generation.

The article has been amended to reflect the correct title.

References

Ushida, T., Cotechini, T., Protopapas, N., Atallah, A., Collyer, C., Toews, A., … Graham, C. (2022). Aberrant inflammation in rat pregnancy leads to cardiometabolic alterations in their offspring and intrauterine growth restriction in the F2 generation. Journal of Developmental Origins of Health and Disease, 1-13. doi: 10.1017/S2040174422000265 CrossRefGoogle Scholar