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Carfentrazone-Ethyl Injury to Spring Wheat (Triticum aestivum) Is Minimized by Some ALS-Inhibiting Herbicides

Published online by Cambridge University Press:  20 January 2017

Kirk A. Howatt*
Affiliation:
Department of Plant Sciences, North Dakota State University, Fargo, ND 58105-5051
*
Corresponding author's E-mail: [email protected]

Abstract

Many herbicides that control broadleaf weeds in wheat can antagonize grass herbicide efficacy, resulting in less grain yield due to competition. Also, the broadleaf herbicide carfentrazone-ethyl can injure wheat, leading to less grain yield because of physiological response. Experiments were conducted to evaluate carfentrazone-ethyl antagonism of fenoxaprop-ethyl, tralkoxydim, clodinafop-propargyl, imazamethabenz, and flucarbazone-sodium and injury to wheat with tank mixes of carfentrazone-ethyl plus each grass herbicide or thifensulfuron, tribenuron, metsulfuron, or imazamox. Carfentrazone-ethyl did not adversely affect control of wild oat or yellow foxtail by grass herbicides in wheat. Carfentrazone-ethyl alone caused 21% wheat injury 3 d after treatment. Adding flucarbazone-sodium to carfentrazone-ethyl resulted in the least wheat injury among treatments that included carfentrazone-ethyl. Fenoxaprop-P-ethyl or clodinafop-propargyl with carfentrazone-ethyl also resulted in less wheat injury compared with carfentrazone-ethyl alone, which was attributed to formulation safeners. Sulfonylurea herbicides provided safening of carfentrazone-ethyl similar to flucarbazone-sodium, while imidazolinones did not alleviate injury. Tralkoxydim plus carfentrazone-ethyl gave 28% wheat injury compared with 21% from carfentrazone-ethyl alone. Wheat injury was no longer visible by 20 d after treatment, and wheat yield was not affected by carfentrazone-ethyl injury when wild oat and yellow foxtail were present or controlled with herbicide.

Type
Research Article
Copyright
Copyright © Weed Science Society of America 

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