Hostname: page-component-586b7cd67f-rcrh6 Total loading time: 0 Render date: 2024-11-22T08:26:25.178Z Has data issue: false hasContentIssue false
  • English
  • Français

The Two-Way Trade—Psychiatry and Neuroscience

Published online by Cambridge University Press:  29 January 2018

Paul R. McHugh  and
Robert G. Robinson
Paul R. McHugh
Affiliation:
School of Medicine, Department of Psychiatry and Behavioral Sciences, Baltimore, Maryland 21205, USA
Robert G. Robinson
Affiliation:
School of Medicine, Department of Psychiatry and Behavioral Sciences, Baltimore, Maryland 21205, USA
Get access Rights & Permissions [Opens in a new window]

Extract

As scientific technologies become more sophisticated, clinicians may come to think that they must devolve into passive recipients rather than contributors to the advances in basic knowledge. This perception and accompanying modesty towards the neurosciences, however, is an error. The intellectual trade between clinician and bench scientist has always been bi-directional and nowhere is this more clearly evident than in recent interactions between psychiatry and neuroscience.

Type
Comments
Information
The British Journal of Psychiatry , Volume 143 , Issue 3 , September 1983 , pp. 303 - 305
Copyright
Copyright © 1983 The Royal College of Psychiatrists 

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

Bunney, W. E. & Davis, J. (1965) Norepinephrine in depressive reactions: A review of supporting evidence. Archives of General Psychiatry, 13, 483–97.CrossRefGoogle Scholar
Folstein, M. F. & McHugh, P. R. (1978) Dementia syndrome of depression. In Alzheimer's Disease: Senile Dementia and Related Disorders (eds. Katzman, R., Terry, R. D. and Bick, K. L.). New York: Raven Press.Google Scholar
Kiloh, L. G. (1961) Pseudo-dementia. Acta Psychiatrica Scandinavica, 37, 336–51.CrossRefGoogle ScholarPubMed
McHugh, P. R. & Folstein, M. F. (1978) Psychopathology of dementia: Implications for neuropathology. Research Publications of Association for Nervous and Mental Disease, 57, 17–30.Google Scholar
Reis, D. J., Ross, R. A., Gilad, G. & Joh, T. H. (1978) Reaction of central catecholaminergic neurons to injury: Model systems for studying the neurobiology of central regeneration and sprouting. In Neuronal Plasticity (ed. Cotman, C. W.). New York: Raven Press.Google Scholar
Robinson, R. G., Shoemaker, W. J., Schlumpf, M., Valk, T. & Bloom, F. E. (1975) Experimental cerebral infarction in rat brain: Effect on catecholamines and behavior. Nature, 255, 332–4.CrossRefGoogle Scholar
Robinson, R. G., Shoemaker, W. J., Schlumpf, M. (1980) Time course of changes in catecholamines following right hemispheric cerebral infarction in the rat. Brain Research, 181, 202–8.CrossRefGoogle ScholarPubMed
Robinson, R. G. & Szetela, B. (1981) Mood change following left hemispheric brain injury. Annals of Neurology, 9, 447–53.CrossRefGoogle ScholarPubMed
Robinson, R. G. & Price, T. R. (1982) Post-stroke depressive disorders: A follow-up study of 103 outpatients. Stroke, 13, 635–41.CrossRefGoogle Scholar
Robinson, R. G., Starr, L. B., Kubos, K. L. & Price, T. R. (1983) A two-year longitudinal study of post-stroke mood disorders: Findings during the initial evaluation. Stroke. In Press.CrossRefGoogle Scholar
Submit a response

eLetters

No eLetters have been published for this article.