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The Significance of Brain Damage in Persistent Oral Dyskinesia

Published online by Cambridge University Press:  29 January 2018

H. Edwards
H. Edwards*
Affiliation:
Department of Psychological Medicine, The Welsh National School of Medicine, Whitchurch Hospital, Cardiff
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Extract

In the last ten years there have been a number of studies reporting an association between certain persistent oral dyskinesias in elderly people (particularly women) and prolonged medication with phenothiazines. Many of the cases described in these studies also showed some evidence of brain damage and because of this it has often been suggested that brain damage is an important contributory cause of oral dyskinesia. Buccal factors such as the edentulous state have also been incriminated (Joyston-Bechal, 1965; Evans, 1965). However, very few controlled studies have been carried out. The literature on this subject has been well reviewed by Crane (1968) and Kline (1968).

Type
Research Article
Information
The British Journal of Psychiatry , Volume 116 , Issue 532 , March 1970 , pp. 271 - 275
Copyright
Copyright © Royal College of Psychiatrists, 1970 

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References

Crane, G. E. (1968). “Tardive dyskinesia in patients treated with major neuroleptics: a review of the literature.” Amer. J. Psychiat., 124, Supp. 40-8.CrossRefGoogle Scholar
Evans, J. H. (1965). “Persistent oral dyskinesia in treatment with phenothiazine derivatives.” Lancet, 458–60.Google Scholar
Hollister, L. E. (1961). “Current concepts in therapy: complications from psychotherapeutic drugs.” New Eng. J. Med., 264, 292.Google Scholar
Hunter, R., Earl, C. J., and Janz, D. (1964). “A syndrome of abnormal movements and dementia in leucotomized patients treated with phenothiazines.” J. Neurol. Neurosurg. Psychiat., 27, 219–23.Google Scholar
Joyston-Bechal, M. P. (1965). “Persistent oral dyskinesia in treatment with phenothiazine derivatives.” Lancet, i, 600–1.Google Scholar
Kline, N. S. (1968). “On the rarity of ‘irreversible’ oral dyskinesias following phenothiazines.” Amer. J. Psychiat., 124, Supp. 48-54.Google Scholar
Pryce, I. G., and Edwards, H. (1966). “Persistent oral dyskinesia in female mental hospital patients.” Psychiat., 112, 983–7.Google Scholar
Shapiro, M. B., Post, F., Lofrino, B., and Inglis, J. (1956). “Memory function in psychiatric patients over sixty, some methodological and diagnostic implications.” J. ment. Set., 102, 233–46.Google Scholar
Walton, D., White, J. G., and Black, D. A. (1959). “A modified word-learning test: a cross-validation study.” Brit. J. med. Psychol., 32, 213–20.Google Scholar
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