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A Neuroendocrine View of ECT
Published online by Cambridge University Press: 02 January 2018
Abstract
“A neuroendocrine hypothesis to explain the clinical activity of convulsive therapy is described. The hypothesis is based on the diabetes/insulin model and suggests that hypothalamic dysfunction with an insuffiency of a mood-maintaining peptide is the basis for affective disorders. Repeated seizures enhance the production and release of the hypothalamic peptide antidepressin – the active principle that relieves both neuroendocrine and behavioral abnormalities.”
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- Copyright © The Royal College of Psychiatrists
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