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Clinical Signs with Modified Electroconvulsive Therapy

Published online by Cambridge University Press:  29 January 2018

J. G. Whitwam ,
T. Moreton  and
J. Norman
J. G. Whitwam
Affiliation:
Department of Anaesthetics, General Infirmary at Leeds
T. Moreton
Affiliation:
Department of Psychiatry, University of Leeds
J. Norman
Affiliation:
Department of Anaesthetics, The General Infirmary at Leeds
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Extract

In many electroconvulsive therapy centres atropine is used to mitigate adverse effects resulting from stimulation of the parasympathetic nervous system, while the violence of muscular contractions is controlled with a muscle relaxant. It is customary to secure hypnosis with a short acting barbiturate prior to administration of the relaxant. The first report of the use of thiopentone for E.C.T. was by Rubinstein (1945), and methohexitone (Lilly 25398) was introduced by Friedman (1959). The use of succinylcholine (iodide) for E.C.T. was first reported by Holmberg and Thesleff (1951). The neurological signs during unmodified E.C.T. have been well documented (e.g. Klein and Early, 1948a, 1948b, 1949; Kalinowsky and Hoch, 1952). There is as yet no comparable detailed clinical description of E.C.T. when modified by atropine and a short acting muscle relaxant preceded by a barbiturate. This discussion is limited to some of those signs which may be of assistance in the management of therapy.

Type
Research Article
Information
The British Journal of Psychiatry , Volume 109 , Issue 460 , May 1963 , pp. 399 - 403
Copyright
Copyright © Royal College of Psychiatrists, 1963

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References

Adderley, D. J., and Hamilton, M., Brit. Med. J., 1953, i, 195.CrossRefGoogle Scholar
Alexander, L., Treatment of Mental Disorders, 1953, 96. Philadelphia and London.Google Scholar
Barron, D. W., and Dundee, J. W., Brit. J. Anaesth., 1962, 34, 90.CrossRefGoogle Scholar
Cerletti, U., and Bini, L., Boll. Acad. Med. Roma., 1938, 64, 36.Google Scholar
Clement, A. J., Brit. Med. J., 1962, i, 228.CrossRefGoogle Scholar
Friedman, E., Dis. nerv. Syst. Monograph Supplement, 1959.Google Scholar
Gold, H., Kwit, N. T., Otto, H., and Fox, T. J., J. Clin. Invest., 1939, 18, 429.CrossRefGoogle Scholar
Holmberg, G., and Thesleff, S., Nord. Med., 1951, 46, 1567.Google Scholar
Kalinowski, L. B., and Hoch, P. H., Shock Treatments, Psychosurgery and other Somatic Treatments in Psychiatry, 1952. New York.Google Scholar
Klein, R., and Early, D. F., J. Ment Sci., 1948a, 94, 581.CrossRefGoogle Scholar
Klein, R., and Early, D. F. ibid., 1948b, 94, 805.Google Scholar
Klein, R., and Early, D. F. ibid., 1949, 95, 638.Google Scholar
Lewis, W. H., Richardson, D. J., and Lawrence, H. G., New Engl. J. Med., 1955, 252, 1016.CrossRefGoogle Scholar
MacMahon, H. E., Amer. J. Pathol., 1929, 5, 333.Google Scholar
Morton, H. J. V., and Thomas, E. T., Lancet, 1958, ii, 1313.CrossRefGoogle Scholar
Nowill, W. K., Wilson, W. P., and Borders, R., Arch. Neurol. Psychiat. (Chicago), 1954, 71, 189.CrossRefGoogle Scholar
Richardson, D. J., Lewis, W. H., Gahagan, L. H., and Sheenan, D., N.Y. St. J. Med., 1957, 57, 881.Google Scholar
Rubinstein, H. S., Science, 1945, 101, 430.CrossRefGoogle Scholar
Thomas, E., and Honan, B. F., Brit. Med. J., 1953, ii, 97.CrossRefGoogle Scholar
Urquhart, R. W. I., J. Indust. Hyg., 1927, 9, 140.Google Scholar
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