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Chromosome 22q11 deletions and aggressive behaviour

Published online by Cambridge University Press:  03 January 2018

H. M. Lachman  and
D. F. Papolos
H. M. Lachman
Affiliation:
Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA
D. F. Papolos
Affiliation:
Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA
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Abstract

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Information
The British Journal of Psychiatry , Volume 172 , Issue 6 , June 1998 , pp. 540
Copyright
Copyright © 1998 The Royal College of Psychiatrists 

References

Lachman, H. Papolos, D. F. Saito, T. et al (1996a) Human catechol-O-methyltransferase pharmacogenetics: description of a functional polymorphism and its potential application to neuropsychiatrie disorders. Pharmacogenetics, 6, 243–250.Google Scholar
Lachman, H. Morrow, B. Shprintzen, R. et al (1996b) Association of codon 108/158 catechol O-methyltransferase gene polymorphism with the psychiatric manifestations of velo-cardio-facial syndrome. American Journal of Medical Genetics (Neuropsychiatric Genetics), 67, 468–472.Google Scholar
Murphy, K. C. Jones, R. G. Griffiths, E. et al (1998) Chromosome 22q11 deletions. An under-recognised cause of idiopathic learning disability. British Journal of Psychiatry, 172, 180–183.Google Scholar
Straus, R. D. Bark, N. Parsia, S. S. et al (1997) Analysis of a functional catechol O-methyltransferase gene polymorphism in schizophrenia: evidence for association with aggressive and antisocial behavior. Psychiatry Research, 69, 71–77.Google Scholar
Volavka, J. (1995) Neurobiology of Violence. Washington, DC: American Psychiatric Press.Google Scholar
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