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Melancholia, Glucose Tolerance and Body Weight

Published online by Cambridge University Press:  08 February 2018

I. G. Pryce
I. G. Pryce*
Affiliation:
Runwell Hospital, near Wickford, Essex
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Extract

A decrease of glucose tolerance in mental illness has been observed for over forty years (Kooy, 1919; Mann, 1925). Although this change in carbohydrate metabolism has been described in all types of mental disorders, the majority of investigators report that it occurs most frequently in melancholia (Kooy, 1919; Mann, 1925; McCowan and Quastel, 1931; Holmgren and Wohlfahrt, 1944). The phenomenon has usually been regarded as a secondary effect, and several theories have been suggested to explain how it is produced (Kooy, 1919; Mann, 1925; McCowan and Quastel, 1931; Holmgren and Wohlfahrt, 1944); however, no single explanation is firmly established, and the mechanism or mechanisms are still uncertain.

Type
Original Articles
Information
Journal of Mental Science , Volume 104 , Issue 435 , April 1958 , pp. 421 - 427
Copyright
Copyright © Royal College of Psychiatrists, 1958 

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References

Chambers, W. H., Physiol. Rev., 1938, 18, 248.Google Scholar
Duncan, L. J. P., Qtly. J. Exp. Phys., 1956, 41, 1, 85.Google Scholar
Epstein, A. A., and Aschner, P. W., J. Biol. Chem., 1916, 25, 151.Google Scholar
Fletcher, A. A., Arch. Int. Med., 1922, 30, 106.Google Scholar
Freeman, H., Rodnick, E. H., Shakow, D., and LeBaux, T., Psychosom. Med., 1944, 6, 4.Google Scholar
Greville, G. D., Biochem. J., 1943, 37, 17.Google Scholar
Idem, Proc. Roy. Soc. Med., 1945, 38, 671.Google Scholar
Halmi, N. S., and Spirtos, B. N., Amer. J. Physiol., 1956, 187, 432.Google Scholar
Henneman, D. H., Altschule, M. D., and Gonez, R. M., Amer. Med. Ass. Arch. Int. Med., 1954, 94, 3.Google Scholar
Holmgren, H., and Wohlfahrt, S., Acta Psychiat. et Neurol., Scand., 1944, Suppl. 31.Google Scholar
Kooy, F. H., Brain, 1919, 42, 214.Google Scholar
Lingjaerde, O., Acta Psychiat. et Neurol. Scand., 1956, Suppl. 106.Google Scholar
McCowan, P. K., and Quastel, J. H., J. Ment. Sci., 1931, 77, 525.CrossRefGoogle Scholar
Mann, S. A., Ibid., 1925, 71, 442.Google Scholar
Rames, E. D., and Simon, W., Arch. Neurol. Psychiat., 1955, 74, 40.Google Scholar
Robinson, G. W., and Shelton, P., J. Amer. M. A., 1940, 114, 2279.Google Scholar
Silverstone, F. A., Brandfonbrener, M., Shock, N. W., and Yiengst, M. J., J. Clin. Invest., 1957, 36, 504.CrossRefGoogle Scholar
Ström-Olsen, R., Lancet, 1932, i, 128.Google Scholar
Weil-Malherbe, K., J. Ment. Sci., 1955, 101, 733.CrossRefGoogle Scholar
Idem and Bone, A. D., ibid., 1951, 97, 635.Google Scholar
Wyshak, G. H., and Chaikoff, I. L., J. Biol. Chem., 1953, 200, 851.Google Scholar
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