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The Effects of Intravenous Insulin in Sakel'S Hypoglycaemic Treatment

Published online by Cambridge University Press:  08 February 2018

A. M. Spencer
A. M. Spencer*
Affiliation:
Joint Counties Mental Hospital, Carmarthen
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Extract

The use of intravenous insulin in Sakel's shock treatment as an alternative to intramuscular insulin was first reported by Ventriglia (1939), whose findings may be summarized as follows:

(1) Hypoglycaemic symptoms appear more quickly than with intramuscular insulin, so that the pre-shock period is shortened.

(2) The coma dose of insulin is reduced by 50 per cent. on an average, but may be reduced by considerably more.

(3) In some cases the hypoglycaemic symptoms are more abrupt and violent.

(4) There is a greater tendency to tonic-clonic spasms and convulsions generally.

Type
Part I.—Original Articles
Information
Journal of Mental Science , Volume 93 , Issue 392 , July 1947 , pp. 483 - 521
Copyright
Copyright © Royal College of Psychiatrists, 1947 

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References

Bardenat, and Ieonardon, (1939), Ann. Med. Psychol., 97 (pt. 1), 220.Google Scholar
Bruger, M., and Friedman, B. (1938), Proc. Soc. Exper. Bio. Med., 38, 840.Google Scholar
Corwin, W. C. (1939), Am. J. Med. Sci., 198, 724.Google Scholar
Cutting, M. (1942), Biochem. J., 36, 376.CrossRefGoogle Scholar
Damashek, W., Myerson, A., and Stephenson, C. (1935), Arch. Neurol. Psychiat., 33, 1.Google Scholar
Finiefs, L. A. (1938), Lancet, 1, 776.CrossRefGoogle Scholar
Fisher, N. F., and Noble, B. E. (1923), Am. J. Physiol., 67, 72.Google Scholar
Freudenberg, R. (1938), Am. J. Psychiat., 94 (Suppl.), 139.CrossRefGoogle Scholar
Freudenberg, R. and Fine, J. (1940), J. Ment. Sci., 86, 84.Google Scholar
Furst, W. (1940), Psychiat. Quart., 14, 397.Google Scholar
Goadby, H. K., and Richardson, J. S. (1940), J. Physiol., 97, 417.Google Scholar
Goldfarb, W. (1943), Am. J. Psychiat., 100, 698.CrossRefGoogle Scholar
Hagedorn, and Jenson, (1923), Biochem. Ztschr., 135, 46; 137, 92.Google Scholar
Hagedorn, H. C. B. N., Jenson, N. B., Krarup, I., and Wodstrup, I. (1936), J. Am. Med. Ass., 106, 177.CrossRefGoogle Scholar
Harrison, G. A. (1943), Chemical Methods in Clinical Pathology, London. Google Scholar
Himsworth, H. P. (1939), Lancet, 2, 1, 65, 118, 171.Google Scholar
Hinko, E. N., Fenton, M. M., and Balberor, H. (1941), Am. J. Psychiat., 98, 140.Google Scholar
Horvath, S. M., and Friedman, E. (1941), J. Clin. Endocrin., 1, 960.Google Scholar
James, G. W. B., Freudenberg, R., and Cannon, A. T. (1938), Proc. Roy. Soc. Med., 31, 578.Google Scholar
Jones, M. (1940), Lancet, 2, 361.CrossRefGoogle Scholar
Kramer, H., and Steiner, A. (1931), Biochem. J., 25, 161.Google Scholar
Longwell, B. B., and Ravin, A. (1936), Am. J. Physiol., 117, 453.CrossRefGoogle Scholar
McGregor, J. S., and Sandison, R. A. (1940), Brit. Med. J., 2, 310.Google Scholar
Mahoney, V. P., and Herskovitz, H. H. (1942), J. Nerv. Ment. Dis., 95, 265.Google Scholar
Nordsted, A., Norgaard, A., and Thaysen, T. E. H. (1930), Acta Med. Sc and., 73, 125.CrossRefGoogle Scholar
Norgaard, A., and Thaysen, T. E. H. (1929), ibid., 72, 492.Google Scholar
Olsen, C. W. (1942), Bull. Los. Ang. Nerv. Soc., 1, 99.Google Scholar
Peterson, M. C., and Lutz, E. H. (1943), J. Nerv. Ment. Dis., 97, 141.Google Scholar
Polatin, P., and Spotnitz, H. (1942a), Am. J. Psychiat., 99, 394.Google Scholar
Polatin, P., and Spotnitz, H. (1942b), Arch. Neurol. Psychiat., 47, 53.Google Scholar
Polatin, P., and Spotnitz, H. (1943), J. Nerv. Ment. Dis., 97, 567.Google Scholar
Polatin, P., Spotnitz, H. and Wiesel, B. (1940a), Arch. Neurol. Psychiat., 43, 925.CrossRefGoogle Scholar
Polatin, P., Spotnitz, H. and Wiesel, B., (1940b), N.Y. State J. Med., 40, 843.Google Scholar
Recordier, M., and Andrac, M. (1935), Marseille Med., 721, 741.Google Scholar
Reznikoff, L., and Scott, J. J. (1942), Psychiat. Quart., 16, 399.Google Scholar
Sakel, M. (1935a), Neue Behandlungmethode der Schizophrenie (Vienna).Google Scholar
Sakel, M. (1935b), The Pharmacological Shock Treatment of Schizophrenia. New York.Google Scholar
Sakel, M. (1938), ibid. Revised Eng. trans. New York.Google Scholar
Sandison, R. A., and McGregor, J. S. (1942), J. Ment. Sci., 88, 387.Google Scholar
Sherman, I. C., Mergener, J. C., and Low, A. A. (1941), Am. J. Psychiat., 98, 77.CrossRefGoogle Scholar
Trevan, J. W. (1941), personal communication.Google Scholar
Trevan, J. W. (1946), personal communication.Google Scholar
Ventriglia, C. (1939), G. Psichiat. Neuropat., 67, 156.Google Scholar
von Angyl, L. (1937), Z. ges. Neurol. Psychiat., 157, 35.Google Scholar
Wilson, I. G. H. (1937), A Study of Hypoglycaemic Shock Treatment in Schizophrenia. His Majesty's Stationery Office.Google Scholar
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