Hostname: page-component-586b7cd67f-2brh9 Total loading time: 0 Render date: 2024-11-25T21:33:51.668Z Has data issue: false hasContentIssue false
  • English
  • Français

Changes in Catechol Amine Response to Successive Electric Convulsive Treatments

Published online by Cambridge University Press:  08 February 2018

Leston L. Havens ,
M. Sheref Zileli ,
Alberto DiMascio ,
Lenore Boling  and
Alan Goldfien
Leston L. Havens
Affiliation:
Harvard Medical School
M. Sheref Zileli
Affiliation:
Harvard Medical School, Peter Bent Brigham Hospital
Alberto DiMascio
Affiliation:
Massachusetts Mental Health Center (Boston Psychopathic Hospital)
Lenore Boling
Affiliation:
Massachusetts Mental Health Center (Boston Psychopathic Hospital)
Alan Goldfien
Affiliation:
Harvard Medical School, Peter Bent Brigham Hospital
Get access Rights & Permissions [Opens in a new window]

Extract

Changes in sympathetic nervous system responses have been noted to accompany changes in psychological states during psychiatric treatment. Funkenstein, Greenblatt and Solomon, observing the blood pressure response to epinephrine and methacholine, have reported a change in blood pressure patterns on the part of patients who benefited from electric shock treatment (1). Gellhorn has presented evidence that improvement in the psychoses is associated with heightened sympathetic reactivity (2).

Type
Original Articles
Information
Journal of Mental Science , Volume 105 , Issue 440 , July 1959 , pp. 821 - 829
Copyright
Copyright © Royal College of Psychiatrists, 1959 

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

1. Funkenstein, D. H., Greenblatt, M., and Solomon, H. C., J. Nerv. and Ment. Dis., 1948, 108, 409.Google Scholar
2. Gellhorn, E., Physiological Foundations of Neurology and Psychiatry, 1953. Minneapolis: University of Minnesota Press.Google Scholar
3. Weil-Malherbe, H., and Bone, A. D., Lancet, 1953, 264, 974.Google Scholar
4. Idem , J. Ment. Sci., 1955, 101, 156.CrossRefGoogle Scholar
5. Goldfien, A., Zileli, M. S., Goodman, D. S., and Thorn, G. W., In preparation.Google Scholar
6. Aronow, L., and Howard, F. A., Federation Proc., 1955, 14, 315.Google Scholar
7. Valk, A. DeT., Jr, ., and Price, H. L., Clin. Invest., 1956, 35, 837.Google Scholar
8. Solomon, A. P., Darrow, C. W., and Blaurock, M., Psychosom. Med., 1939, 1, 118.CrossRefGoogle Scholar
9. Gellhorn, E., and Safford, H., Proc. Soc. Exp. Biol. and Med., 1948, 68, 74.Google Scholar
10. Wenger, M. A., Engel, B. T., and Clemens, T. L., Behavioral Sci., 1957, 2, 216.CrossRefGoogle Scholar
11. Havens, L. L., Dis. Nerv. System, 1958, 19, 29.Google Scholar
12. Michael, S. T., Yale J. Biol. and Med., 1949, 22, 71.Google Scholar
13. Idem , and Brown, W. T., J. Nerv. and Ment. Dis., 1951, 113, 538.Google Scholar
14. Altschule, M. D., Parkhurst, B. H., and Tillotson, K. J., J. Clin. Endocrinol., 1949, 9, 440.Google Scholar
15. Holland, W. C., and Schumann, H. J., Brit. J. Pharmacol., 1956, 11, 449.Google Scholar
16. Zileli, M. S., Walker, W., Reutter, F., Shoemaker, W., and Friend, D. In preparation.Google Scholar
17. Satake, Y., 1955. Tokyo: Nanzando Co., Ltd.Google Scholar
18. Elmadjian, F., and Hope, J. M., Arch. Neurol. and Psychiat., 1957, 78, 38.Google Scholar
19. Von Euler, U. S., and Hellner, S., Acta physiol. scandinav., 1952, 26, 183.Google Scholar
20. Thesleff, S., ibid., 1952, 26, 103.Google Scholar
Submit a response

eLetters

No eLetters have been published for this article.