Hostname: page-component-745bb68f8f-d8cs5 Total loading time: 0 Render date: 2025-01-11T00:10:38.991Z Has data issue: false hasContentIssue false
  • English
  • Français

Periodic Catatonia

Published online by Cambridge University Press:  08 February 2018

J. S. Bonar Lindsay
J. S. Bonar Lindsay*
Affiliation:
Division of Mental Hygiene, Health Department, N.Z.
Get access Rights & Permissions [Opens in a new window]

Extract

Kraepelin (10) in 1908 first defined a group within his classification of schizophrenia which he described as periodic catatonia. The clinical features of this type may be briefly summarized. The onset, usually between 14 and 20 years, is frequently characterized by a period of erratic conduct followed by an interval of stupor, confusion or excitement, which thereafter undergoes periodic remissions and relapses. The periodicity is remarkable in the rapidity with which all changes occur. In the periods of remission the patient is free of catatonia and associated symptom-complex (sympatheticotonia), but is still dull, apathetic and withdrawn; he often recalls events from the reactive phase, but without insight. The patient is still schizophrenic, with emotional flattening and poverty of ideation, and remains so throughout. In the reactive phase the picture is one of catatonic stupor or excitement, with clinical evidence of sympathetic over-activity.

Type
Part I.—Original Articles
Information
Journal of Mental Science , Volume 94 , Issue 396 , July 1948 , pp. 590 - 602
Copyright
Copyright © Royal College of Psychiatrists, 1948 

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

Literature

(1) Alpers, B. J. (1940), “Personality and Emotional Disorders Associated with Hypothalamic Lesions,” Res. Publ. Assn. Nerv. Ment. Disease, 20, 725.Google Scholar
(2) Bailey, P., et al. (1945), “Effects of Lesion of the Periaqueductal Gray Matter in Cats and Monkeys,” Arch. Neurol. and Psychiat., 53, 325.Google Scholar
(3) Boisen, A. T. (1936), The Exploration of the Inner World. New York: Willett, Clark & Co., Chicago.Google Scholar
(4) Cairns, H., et al. (1941), “Akinetic Mutism with an Epidermoid Cyst of the Third Ventricle,” Brain, 64, 273.CrossRefGoogle Scholar
(5) De Jong, H. H. (1945), Experimental Catatonia. Baltimore: The Williams & Wilkins Co.Google Scholar
(6) Furtado, D., et al. (1946), “Catatonia from Diencephalic Compression,” Revue Neurol., 78, 449.Google Scholar
(7) Gjessing, R. (1938), “Disturbances of Somatic Function in Catatonia with a Periodic Course and Their Compensation,” J. Ment. Sci., 84, 608.CrossRefGoogle Scholar
(8) Hoskins, R. G. (1946), The Biology of Schizophrenia. New York: W. W. Norton & Co.Google Scholar
(9) Jantz, H. (1941), “Metabolic Changes during Mescal Intoxication in Man and Animals; Similarity to Metabolic Changes in Schizophrenia,” Ztschr. f. d. ges. Neurol. u. Psychiat., 171, 28.CrossRefGoogle Scholar
(10) Kraepelin, E. (1913), Psychiatrie. Eighth ed. 3, 806.Google Scholar
(11) Oller, C. I. (1946), “Paroxysmal Autonomic Discharges in the Post-encephalitic State,” Arch. Neurol. and Psychiat., 55, 388.CrossRefGoogle Scholar
(12) Ostow, M. (1943), “Recurrent Autonomic Phenomena Associated with Exacerbation of Post-encephalitic Parkinsonism,” ibid., 50, 342.Google Scholar
(13) Penfield, W. (1929), “Diencephalic Autonomic Epilepsy,” ibid., 22, 358.Google Scholar
(14) Pollak, O. J. (1944), “Post-mortem Studies in Mental Patients,” Amer. J. Clin. Pathol., 14, 289.CrossRefGoogle Scholar
(15) Stocks, J. W. (1930), “A Case of Pituitary Cachexia,” Lancet, 2, 349.CrossRefGoogle Scholar
(16) Stokes, A. B. (1941), “Metabolic Investigations in Periodic Catatonia,” Proc. R. Soc. Med., 34, 733.Google Scholar
(17) Idem (1939), “Somatic Research in Periodic Catatonia,” J. Neurol. and Psychiat., 2, 243.CrossRefGoogle Scholar
Submit a response

eLetters

No eLetters have been published for this article.