Hostname: page-component-78c5997874-8bhkd Total loading time: 0 Render date: 2024-11-09T19:50:36.225Z Has data issue: false hasContentIssue false

Part II. The Significance of Urinary Reaction in Psychotic Subjects

Published online by Cambridge University Press:  19 February 2018

Extract

In the normal subject the reaction of the urine denotes the tendency to reaction change of the blood and organism—a tendency which is adjusted by the respiratory mechanisms. To maintain a constant blood pH and CO2/NaHCO3, the kidney retains or rejects base and the lungs increase or decrease acid (C02) output, these compensatory processes being interdependent. With a definite relationship between urinary acidity, blood alkali reserve and alveolar air CO2 tension the ratios are maintained. Urinary reaction represents the acid or alkaline “pressure”on the organism. The reaction of the blood and tissues depends on the adequacy of respiratory adjustment: in fact, as Henderson says, “low pH (acidity) in a fluid buffered like blood can never be due immediately to any other cause than relatively depressed breathing.”

Type
Part I.—Original Articles
Copyright
Copyright © Royal College of Psychiatrists, 1928 

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

(1) Henderson, Y., Physiological Reviews, 1926, v, p. 131.Google Scholar
(2) Hasselbalch, , Biochem. Zeit., 1912, xlvi, p. 403.Google Scholar
(3) Beckmann, , Zeit. f. d. ges. Exper. Med., 1922, xxix, p. 579; and 1924, xlii, p. 424.Google Scholar
(4) [Mann, and Scott, , Mott Memorial Volume. Google Scholar
(5) Leathes, , Brit. Med. Journ., 1919, ii, p. 165.Google Scholar
(6) Endres, , Biochem. Zeit., 1922, cxxxii, p. 220.Google Scholar
(7) Idem, ibid., 1923, cxlii, p. 53.Google Scholar
(8) Straub, , Deutsch. Arch. klin. Med., 1915, cxvii, p. 397.Google Scholar
(9) Davies, , Haldane, and Kennaway, , Journ. Physiol., 1920, liv, p. 32.CrossRefGoogle Scholar
(10) Henderson, and Haggard, , Journ. Biol. Chem., 1918, xxxiii, p. 333.CrossRefGoogle Scholar
(11) Simpson, and Wells, , ibid., 1928, lxxvi, p. 171.Google Scholar
(12) Bazett, and Haldane, , Proc. Physiol. Soc., 1925, xlv, p. 4.Google Scholar
(13) Koehler, , Arch. Int. Med., 1923, xxxi, p. 59.Google Scholar
(14) Haldane, J. S., Respiration, Newhaven, 1922.CrossRefGoogle Scholar
(15) Mann, , Morris, and Rowe, , Journ. Ment. Sci., 1928, lxxiv, p. 425.CrossRefGoogle Scholar
(16) Marriot, and Howland, , Arch. Int. Med., 1916, xviii, p. 788.Google Scholar
(17) De Wesselow, , Chemistry of the Blood in Clinical Medicine, London, 1924.Google Scholar
(18) Herrmannsdorfer, , Jung, and Stein, , Münch. med. Woch., 1927, lxxiv, p. 711.Google Scholar
(19) Gamble, , Ross, and Tisdall, , Journ. Biol. Chem., 1923, lvii, p. 633.Google Scholar
(20) Palmer, and Van Slyke, , ibid., 1917, xxxii, p. 499.Google Scholar
(21) Palmer, , Salveson, and Jackson, jun., ibid., 1921, xlv, p. 101.Google Scholar
(22) Robertson, I. M., Mott Memorial Volume. Google Scholar
Submit a response

eLetters

No eLetters have been published for this article.