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Published online by Cambridge University Press: 12 September 2005
in the past few years, the biochemistry of the ataxia–telangiectasia mutated (atm) protein and our knowledge of its cellular functions has surged ahead. the neurology of ataxia–telangiectasia (at), including its presenting features in early childhood, as well as its progression and further ramifications in early adulthood, were discussed at the international conference on at in birmingham, uk.