Hostname: page-component-586b7cd67f-r5fsc Total loading time: 0 Render date: 2024-11-28T16:16:55.109Z Has data issue: false hasContentIssue false
  • English
  • Français

Graves' Thyrotoxicosis and Moyamoya Disease

Published online by Cambridge University Press:  18 September 2015

Kenji Kushima ,
Yutaka Satoh ,
Yoshio Ban ,
Matsuo Taniyama ,
Kunihiko Ito  and
Koujiro Sugita
Kenji Kushima*
Affiliation:
Third Department of Internal Medicine, Tokyo
Yutaka Satoh
Affiliation:
Department of Neurology, Tokyo
Yoshio Ban
Affiliation:
Third Department of Internal Medicine, Tokyo
Matsuo Taniyama
Affiliation:
Third Department of Internal Medicine, Tokyo
Kunihiko Ito
Affiliation:
Showa University School of Medicine, Ito Hospital, Tokyo
Koujiro Sugita*
Affiliation:
Department of Neurology, Tokyo
*
3rd Dept. of Internal Medicine, Showa University School of Medicine, 1-5-8 Hatanodai Shinagawa-ku Tokyo, Japan (142)
3rd Dept. of Internal Medicine, Showa University School of Medicine, 1-5-8 Hatanodai Shinagawa-ku Tokyo, Japan (142)
Rights & Permissions [Opens in a new window]

Abstract:

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

Cerebrovascular accidents due to Moyamoya disease, a disorder characterized by arterial stenosis at the base of the brain accompanied by typical net-like collateral vessels, occurred in two young Japanese women with Graves' disease when they were in thyrotoxicosis. In one patient, a second attack of cerebral infarction occurred with the recurrence of thyrotoxicosis. Association of Moyamoya disease and Graves' thyrotoxicosis is rare and the pathegenetic relationship is discussed.

Type
Original Articles
Information
Canadian Journal of Neurological Sciences , Volume 18 , Issue 2 , May 1991 , pp. 140 - 142
Copyright
Copyright © Canadian Neurological Sciences Federation 1991

References

1.Suzuki, J, Tanaka, A. Cerebrovascular “Moyamoya” disease. Disease showing abnormal net-like vessels in base of brain. Arch Neurol 1969; 20: 288–299.CrossRefGoogle ScholarPubMed
2.Suzuki, J, Kodama, M, Fujiwara, S, et al. A study about the cause of Moyamoya disease. The 1980 Annual Report of the Research Committee on the Idiopathic Progressive Occlusive Disease of Circle of Willis; Tokyo, Japanese Ministry of Health and Welfare, 1981: 21–34.Google Scholar
3.Ingbar, SH. The Thyroid Gland. In: Wilson, JD, Foster, DW, eds. Textbook of Endocrinology. Philadelphia: Saunders, 7th Ed. 1985; 682–815.Google Scholar
4.Suzuki, J, Kowada, M, Asahi, M, et al. A study of disease showing singular cerebral angiographic findings which seems to be new collateral circulation. Proc. 22nd Meet Jap Neurosurg Soc 1963.Google Scholar
5.Handa, H. A summarized report. The 1987 Annual Report of the Research Committee on the Idiopathic Progressive Occlusive Disease of Circle of Willis; Tokyo, Japanese Ministry of Health and Welfare 1988: 1–4.Google Scholar
6.Matsubayashi, K, Fukuyama, H, Akiguchi, I, et al. Re-evaluation of hypoplasia of bilateral internal carotid arteries in young adults — Association with abnormal thyroid function. Proc 24th Meel Jap Neurol Soc 1983.Google Scholar
7.Sokoloff, L, Wechsler, RL, Mangold, R, et al. Cerebral blood flow and oxygen consumption in hyperthyrodism before and after treatment. J Clin Invest 1953; 32: 202–208.CrossRefGoogle Scholar