
Book contents
- Frontmatter
- Contents
- List of contributors
- Preface
- Section 1 Cardiac pseudotumors and other challenging diagnoses
- Section 2 Cardiac aneurysms and diverticula
- Section 3 Anatomic variants and congenital lesions
- Section 4 Coronary arteries
- Section 5 Pulmonary arteries
- Section 6 Cardiovascular MRI artifacts
- Section 7 Acute aorta and aortic aneurysms
- Section 8 Post-operative aorta
- Section 9 Mesenteric vascular
- Case 73 Pseudostenosis of the common bile duct from crossing hepatic artery
- Case 74 Pseudometastatic disease from hepatic arterioportal shunts
- Case 75 Pancreatic pseudomass due to thrombosed pseudoaneurysm
- Case 76 Splenic artery aneurysm mimicking pancreatic neuroendocrine tumor
- Case 77 Median arcuate ligament compression
- Case 78 Non-occlusive mesenteric ischemia
- Case 79 Segmental arterial mediolysis
- Case 80 Superior mesenteric artery syndrome
- Case 81 Renal fibromuscular dysplasia
- Case 82 Reversal of superior mesenteric artery and vein in midgut volvulus
- Case 83 Mesenteric artery collateral pathways
- Case 84 Mesenteric artery anatomic variants
- Section 10 Peripheral vascular
- Section 11 Veins
- Index
- References
Case 80 - Superior mesenteric artery syndrome
from Section 9 - Mesenteric vascular
Published online by Cambridge University Press: 05 June 2015
- Frontmatter
- Contents
- List of contributors
- Preface
- Section 1 Cardiac pseudotumors and other challenging diagnoses
- Section 2 Cardiac aneurysms and diverticula
- Section 3 Anatomic variants and congenital lesions
- Section 4 Coronary arteries
- Section 5 Pulmonary arteries
- Section 6 Cardiovascular MRI artifacts
- Section 7 Acute aorta and aortic aneurysms
- Section 8 Post-operative aorta
- Section 9 Mesenteric vascular
- Case 73 Pseudostenosis of the common bile duct from crossing hepatic artery
- Case 74 Pseudometastatic disease from hepatic arterioportal shunts
- Case 75 Pancreatic pseudomass due to thrombosed pseudoaneurysm
- Case 76 Splenic artery aneurysm mimicking pancreatic neuroendocrine tumor
- Case 77 Median arcuate ligament compression
- Case 78 Non-occlusive mesenteric ischemia
- Case 79 Segmental arterial mediolysis
- Case 80 Superior mesenteric artery syndrome
- Case 81 Renal fibromuscular dysplasia
- Case 82 Reversal of superior mesenteric artery and vein in midgut volvulus
- Case 83 Mesenteric artery collateral pathways
- Case 84 Mesenteric artery anatomic variants
- Section 10 Peripheral vascular
- Section 11 Veins
- Index
- References
Summary
Imaging description
Superior mesenteric artery syndrome is usually evaluated by contrast-enhanced CT or magnetic resonance angiography (MRA). The classic imaging finding is reduced space between the superior mesenteric artery and the anterior wall of the abdominal aorta that results in duodenal narrowing. The aortomesenteric angle, obtained from sagittal images, is significantly reduced from the normal range of 38–65°. The combination of an aortomesenteric angle less than 22° and an aortomesenteric distance of less than 8–10 mm is considered by some authors to meet criteria for the diagnosis of SMA syndrome in the right clinical setting on CT (Figure 80.1). Additional supportive findings include minimal intra-abdominal and retroperitoneal fat, duodenal compression between the aorta and SMA, dilation of the first and second portions of the duodenum, left renal vein enlargement, and enlargement of the left gonadal vein or other venous collaterals as a result of chronic renal vein compression (Figure 80.1).
Importance
Young age and non-specific symptoms often lead to a delay in diagnosis, resulting in complications such as malnutrition, dehydration, and electrolyte abnormalities in patients with SMA syndrome.
Typical clinical scenario
Superior mesenteric artery (SMA) syndrome is an atypical cause of high intestinal obstruction, with estimated incidence rates based on gastrointestinal barium series from 0.01% to 0.33%. It occurs from an abnormally short distance between the aorta and SMA from loss of intra-abdominal fat, which normally separates them resulting in duodenal compression and is an important differential in patients with postprandial abdominal pain, vomiting, and weight loss. The most common predisposing factors include severe weight loss and cachexia, surgical correction of spinal deformities, and congenital anomalies. Patients with recent bariatric surgery, cancer, or chronic immobilization are at risk.
Common symptoms include intermittent epigastric pain, which is often postprandial, early satiety, nausea, fullness, and voluminous vomiting, most frequently occurring in patients who have experienced rapid weight loss.
- Type
- Chapter
- Information
- Pearls and Pitfalls in Cardiovascular ImagingPseudolesions, Artifacts, and Other Difficult Diagnoses, pp. 250 - 251Publisher: Cambridge University PressPrint publication year: 2015