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2.53 - Transdermal Selegiline

from Monoamine Oxidase Inhibitor Antidepressants

Published online by Cambridge University Press:  19 October 2021

Edited by
Michael Cummings  and
Stephen Stahl
Michael Cummings
Affiliation:
University of California, Los Angeles
Stephen Stahl
Affiliation:
University of California, San Diego
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Summary

Typical or first-generation antipsychotic (FGA)

Type
Chapter
Information
Management of Complex Treatment-resistant Psychotic Disorders , pp. 338 - 341
Publisher: Cambridge University Press
Print publication year: 2021

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References

Shulman, K. I., Herrmann, N., Walker, S. E. (2013). Current place of monoamine oxidase inhibitors in the treatment of depression. CNS Drugs, 27, 789–797.CrossRefGoogle ScholarPubMed
Lee, K. C., Chen, J. J. (2007). Transdermal selegiline for the treatment of major depressive disorder. Neuropsychiatr Dis Treat, 3, 527–537.Google ScholarPubMed
Clarke, A., Brewer, F., Johnson, E. S., et al. (2003). A new formulation of selegiline: improved bioavailability and selectivity for MAO-B inhibition. J Neural Transm (Vienna), 110, 1241–1255.CrossRefGoogle ScholarPubMed
Goodnick, P. J. (2007). Seligiline transdermal system in depression. Expert Opin Pharmacother, 8, 59–64.Google Scholar
Patkar, A. A., Pae, C. U., Masand, P. S. (2006). Transdermal selegiline: the new generation of monoamine oxidase inhibitors. CNS Spectr, 11, 363–375.CrossRefGoogle ScholarPubMed
Blob, L. F., Sharoky, M., Campbell, B. J., et al. (2007). Effects of a tyramine-enriched meal on blood pressure response in healthy male volunteers treated with selegiline transdermal system 6 mg/24 hour. CNS Spectr, 12, 25–34.CrossRefGoogle ScholarPubMed

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