Published online by Cambridge University Press: 08 September 2022
The focus of this chapter is on evolutionary theories and models of anorexia nervosa (AN), bulimia nervosa (BN) and obesity. Although obesity is not considered a mental health problem, its link with binge eating disorder and its massively increased prevalence in recent decades, in association with modernisation and Westernisation together with increased morbidity and mortality, have stimulated much evolutionary theorising. Disorders of eating and weight are of particular interest to evolutionary scholars for a number of reasons. These include the claim that many of these disorders are evolutionarily novel, that they have increased in prevalence in developed countries in recent decades, that they have a large female preponderance, particularly of AN and BN, and that they have an increased risk of mortality. Our poor understanding of the aetiology of eating disorders together with poor outcomes (especially for AN) has been associated with a proliferation of proximate theories/models within mainstream psychiatry but without any one theory gaining wide acceptance. This presents an opportunity for evolutionary models to propose new ways of thinking and new avenues for research on these disorders. A review of the current evolutionary literature on AN and BN shows that despite the wide range and variety of models, the sexual competition hypothesis has, so far, had the strongest empirical support from clinical and non-clinical studies. While other evolutionary theories focus on AN, the sexual competition hypothesis provides an explanation for both AN and BN, as well as for the widespread dieting seen in the population. Furthermore, it uniquely makes sense of the specific presentations of eating disorders in males. Nevertheless, it seems increasingly clear that intrasexual competition is not the whole story. More recent work that considers other areas of mismatch in the modern environment represents a necessary extension to this theoretical perspective. It is concluded that larger-scale studies on clinical populations are required to put these theoretical formulations to the test and to explore their potential clinical utility.
To save this book to your Kindle, first ensure [email protected] is added to your Approved Personal Document E-mail List under your Personal Document Settings on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part of your Kindle email address below. Find out more about saving to your Kindle.
Note you can select to save to either the @free.kindle.com or @kindle.com variations. ‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi. ‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
Find out more about the Kindle Personal Document Service.
To save content items to your account, please confirm that you agree to abide by our usage policies. If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account. Find out more about saving content to Dropbox.
To save content items to your account, please confirm that you agree to abide by our usage policies. If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account. Find out more about saving content to Google Drive.