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2 - Preoperative Instructions and Intraoperative Environment

from PART I - MINIMALLY INVASIVE ANESTHESIA (MIA)Ⓡ FOR MINIMALLY INVASIVE SURGERY

Published online by Cambridge University Press:  22 August 2009

Barry L. Friedberg M.D.
Affiliation:
Assistant Professor in Clinical Anesthesia, Keck School of Medicine, University of Southern California
Barry Friedberg
Affiliation:
Keck School of Medicine, University of Southern California
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Summary

PREOPERATIVE INSTRUCTIONS

Nothing per os (NPO), or nothing by mouth, after midnight is the most commonly given preoperative instruction to all surgical patients. This is not unreasonable given the fact that the majority of surgical patients are exposed to emetogenic inhalational vapors and/or emetogenic intravenous opioids. Both inhalational vapors and intravenous opioids depress the laryngeal or “life-protecting” reflexes.

California Assembly Bill (AB)595 specifically mandated office accreditation when sedatives and analgesics are used in a manner that has the probability to depress the “life-preserving” reflexes. The “Catch-22” is that neither the legislature nor the anesthesia community ever defined what the “life-preserving” reflexes are. In both the peer-reviewed literature1 and in unrebutted public testimony before theCAMedical Board when Dr. Thomas Joas, a prominent anesthesiologist, was its presiding chairman, Friedberg has unequivocally defined the laryngeal reflexes as the “life-preserving” reflexes.

Emetogenic inhalational vapors and/or emetogenic intravenous opioids expose the surgical patient to an increased likelihood of aspiration and death. If surgical patients cannot reflexly protect their trachea, they cannot preserve their lives. The lack of pharyngeal reflexes or swallowing seen with propofol sedation/anesthesia does not necessarily mean that the laryngeal reflexes are similarly depressed. In fact, when ketamine is added to the regimen of propofol sedation and opioids are scrupulously avoided (i.e., PK MAC/MIA™ technique), laryngospasm has been observed in about 1–2% of patients. Laryngospasm is the antithesis of depressed laryngeal reflexes.

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Chapter
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Publisher: Cambridge University Press
Print publication year: 2007

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References

Friedberg, BL: Propofol-ketamine technique, dissociative anesthesia for office surgery: A five-year review of 1,264 cases. Aesth Plast Surg 23:70, 1999.Google Scholar
Iverson, R. E., Lynch, D. J., and the ASPS Committee on Patient Safety: Practice advisory on liposuction. Plast Reconstr Surg 100:1478, 2004.Google Scholar
Friedberg, BL: Inaccuracies and omissions with the report of the ASPS Committee on Patient Safety Practice Advisory on Liposuction. Plast Reconstr Surg 117:2142, 2005.Google Scholar
Corssen, G, Miyasaka, M, Domino, EF: Changing concepts in pain control during surgery: Dissociative anesthesia with CI-581. Anesth Analg Curr Res 47:746, 1968.Google Scholar
Court, MH, Duan, SX, Hesse, LM, et al.: Cytochrome P-450 2B6 is responsible for interindividual variability of propofol hydroxylation by human liver microsomes. Anesthesiol 94:110, 2001.Google Scholar
Gan, TJ, Meyer, T, Apfel, CC, et. al.: Consensus guidelines for managing postoperative nausea and vomiting. Anesth Analg 97:62, 2003.Google Scholar
Apfel, CC, Korttila, K, Abdalla, M, et al.: A factorial trial of six interventions for the prevention of PONV. N Engl J Med 350:2441, 2004.Google Scholar
Friedberg BL: Propofol ketamine anesthesia for cosmetic surgery in the office suite, in Osborne, I (ed.), Anesthesia for Outside the Operating Room. International Anesthesiology Clinics. Baltimore, Lippincott, Williams & Wilkins, 41:39, 2003.
Macario, A, Weinger, M, Carney, K, et al.: Which clinical anesthesia outcomes are important to avoid? The perspective of patients. Anesth Analg 89:652, 1999.Google Scholar
Buxton, D: Anaesthetics. London, H.K. Lewis, 1888; p 145.
Balasubramaniam, B, Park, GR: Sexual hallucinations during and after sedation and anaesthesia. Anaesthesia 58:1149, 2003.Google Scholar

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