Skip to main content Accessibility help

We use cookies to distinguish you from other users and to provide you with a better experience on our websites. Close this message to accept cookies or find out how to manage your cookie settings.

Close cookie message
×
  1. Home
  2. Search

Refine search

Refine search

Access:
Content type:
Publication date:
Apply   From and To filters
Subject: Show more
Journals Show more
Publishers: Show more
Societies Show more
Series: Show more
Collections: Show more

Actions for selected content:

Select all | Deselect all
  • View selected items
  • Export citations
  • Download PDF (zip)
  • Save to Kindle
  • Save to Dropbox
  • Save to Google Drive

Save Search

You can save your searches here and later view and run them again in "My saved searches".

Please provide a title, maximum of 40 characters.
Cancel
×

1 results

  • 21 - Fluid and Hemodynamic Monitoring in Burns

  • from Section 3 - Practical Use
    • By
  • Edited by Alexandre Joosten, University of California, Los Angeles, Maxime Cannesson, University of California, Los Angeles, Robert G. Hahn, Karolinska Institutet, Stockholm
  • Book:
    Hemodynamic Monitoring and Fluid Therapy during Surgery
    Published online:
    11 April 2024
    Print publication:
    02 May 2024, pp 249-256

  • Summary

    Severe burn injury induces an early and profound hypovolemia, rapidly followed by a systemic inflammatory response syndrome (SIRS) resulting in a distributive shock.

    Cardiovascular consequences of severe burn injury are multiple including burn edema, burn shock, burn-associated cardiac injury and alteration of microcirculation

    Hemodynamic targets of critically ill burn patients and goal-directed resuscitation therapy are the cornerstone of initial hemodynamic management.

    This resuscitation is challenging with the risk of under- and over-resuscitation justifying an invasive hemodynamic monitoring.

    Balanced crystalloids are the most commonly used fluids in severely burned patients; the use of albumin is controversial.

    During the distributive phase, norepinephrine is often required 12 to 36 hours post-injury.