Previous research has shown that haloxyfop {2-[4-[[3-chloro-5-(trifluoromethyl)-2-pyridinyl] oxy] phenoxy] propanoic acid} and sethoxydim {2-[1-(ethoxyimino)butyl-5-[2-(ethylthio)propyl]-3-hydroxy-2-cyclohexen-1-one} give less control of quackgrass [Agropyron repens (L.) Beauv. # AGRRE] when applied at the five- to seven-leaf stage compared to the three- to four-leaf stage. Current research indicates that reduced control of quackgrass in the eight-leaf stage, compared to the four-leaf stage, was not due to reduced herbicide retention on leaves or to changes in residual herbicide activity in the soil following postemergence applications. The efficacy of both herbicides on rhizome bud kill did not change between the four- and eight-leaf stages following application to the foliage. However, the efficacy of both herbicides on crown tissue bud viability was less at the eight-leaf stage. Rhizome sink intensity (the capacity to accumulate radiolabel) was similar at both stages of development following 14C-haloxyfop and 14C-sethoxydim application to either upper or lower leaves. Crown tissue sink intensity was significantly less at the eight-leaf stage compared to the four-leaf stage following 14C-sethoxydim application to either upper or lower leaves and following 14C-haloxyfop application to lower leaves. These data suggest that crown bud regrowth contributes substantially to reduced control of quackgrass in advanced stages of growth following applications of haloxyfop and sethoxydim.