Book contents
- Frontmatter
- Contents
- Preface
- Acknowledgements
- Section 1 Diaphragm and adjacent structures
- Section 2 Liver
- Section 3 Biliary system
- Section 4 Spleen
- Case 27 Pseudofluid due to complete splenic infarction
- Case 28 Pseudosubcapsular hematoma
- Case 29 Splenic hemangioma
- Case 30 Littoral cell angioma
- Section 5 Pancreas
- Section 6 Adrenal glands
- Section 7 Kidneys
- Section 8 Retroperitoneum
- Section 9 Gastrointestinal tract
- Section 10 Peritoneal cavity
- Section 11 Ovaries
- Section 12 Uterus and vagina
- Section 13 Bladder
- Section 14 Pelvic soft tissues
- Section 15 Groin
- Section 16 Bone
- Index
- References
Case 29 - Splenic hemangioma
from Section 4 - Spleen
Published online by Cambridge University Press: 05 November 2011
- Frontmatter
- Contents
- Preface
- Acknowledgements
- Section 1 Diaphragm and adjacent structures
- Section 2 Liver
- Section 3 Biliary system
- Section 4 Spleen
- Case 27 Pseudofluid due to complete splenic infarction
- Case 28 Pseudosubcapsular hematoma
- Case 29 Splenic hemangioma
- Case 30 Littoral cell angioma
- Section 5 Pancreas
- Section 6 Adrenal glands
- Section 7 Kidneys
- Section 8 Retroperitoneum
- Section 9 Gastrointestinal tract
- Section 10 Peritoneal cavity
- Section 11 Ovaries
- Section 12 Uterus and vagina
- Section 13 Bladder
- Section 14 Pelvic soft tissues
- Section 15 Groin
- Section 16 Bone
- Index
- References
Summary
Imaging description
Splenic hemangiomas are non-encapsulated benign proliferations of vascular channels that range from capillary to cavernous in size [1]. Smaller hemangiomas are usually solid while larger hemangiomas may be partially cystic or calcified, presumably secondary to thrombosis and infarction (Figures 29.1 and 29.2). At ultrasound, splenic hemangiomas typically appear as well-defined solid echogenic or complex partially cystic masses. At CT, hemangiomas appear as hypodense well-circumscribed masses with marked homogeneous enhancement of the solid components. Delayed enhancement has been described for splenic hemangiomas at CT, but they reportedly have a mottled heterogeneous appearance in contrast to the typical centripetal enhancement of hepatic hemangiomas [2]. At MRI, smaller splenic hemangiomas resemble hepatic hemangiomas with T2 hyperintensity and delayed centripetal enhancement [3], but larger lesions are more variable [1]. There is some discrepancy between the reported CT versus MRI delayed enhancement patterns [2, 3], but this may partially reflect the fact that routine MRI includes more delayed acquisition times than routine CT. While splenic hemangiomas may demonstrate centripetal enhancement (Figures 29.3 and 29.4), they lack the typical nodular globular pattern of peripheral enhancement seen in hepatic hemangiomas [1, 4]. This may reflect differences in vascular supply (dual blood supply to the liver versus single blood supply to the spleen), but this is speculative.
- Type
- Chapter
- Information
- Pearls and Pitfalls in Abdominal ImagingPseudotumors, Variants and Other Difficult Diagnoses, pp. 98 - 101Publisher: Cambridge University PressPrint publication year: 2010